The lymphatic vasculature in disease
By K. Alitalo. Nature Medicine, 2011
932 citations recorded by the source corpus.
Subfields: Molecular Biology, Oncology, Pathology and Forensic Medicine
View Paper: https://www.semanticscholar.org/paper/448d7799391bfbe999a05e7df7fedb09c53a893c
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Citations in the corpus
- Interstitial fluid and lymph formation and transport: physiological regulation and roles in inflammation and cancer. (2012, background): “Since our focus is lymph formation and transport, we will not cover the molecular regulation of lymphangiogenesis and lymphatic function but rather refer to excellent recent reviews addressing these topics (11, 12, 222, 305, 459), along with a review of how the interstitial microenvironment can infl”
- Normalizing tumor microenvironment to treat cancer: bench to bedside to biomarkers. (2013, background): “homeostasis in tissues but also in immune response and lymphatic metastasis.(124) To this end, we began investigating lymphatic function”
- The tumor microenvironment at a glance (2012, background): “Tumors drive lymphangiogenesis or lymphatic hyperplasia through the production of VEGFC or VEGFD (Alitalo, 2011).”
- Revisiting the concept of CNS immune privilege (2015, background): “In peripheral tissues, circulating ISF is recycled into the local lymphatic system, thereby cleaning the tissue and preventing the accumulation of potentially toxic compounds such as cellular metabolites or debris from damaged or necrotic cells [27].”
- CCBE1 Enhances Lymphangiogenesis via A Disintegrin and Metalloprotease With Thrombospondin Motifs-3–Mediated Vascular Endothelial Growth Factor-C Activation (2014, background): “1962 Vascular endothelial growth factor (VEGF) -C is the main driver of lymphangiogenesis in embryonic development and in various lymphangiogenic processes in adults.(1) It acts via VEGF receptor (VEGFR) -3 and, in its proteolytically processed mature form, also via VEGFR-2.”