Neurobiology of emotion perception II: Implications for major psychiatric disorders.
By Mary L. Phillips, Wayne C. Drevets, Scott L. Rauch, Richard D. Lane. Biological Psychiatry, 2003
1,768 citations recorded by the source corpus.
Subfields: Cognitive Neuroscience, Experimental and Cognitive Psychology, Psychiatry and Mental health
View Paper: https://www.semanticscholar.org/paper/f50e77af06b17beda32761eb8963fdba9f3c52b6
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Cognitive Neuroscience
- James J. Gross
- Kevin N. Ochsner
- A. Beck
- Paul Gilbert
- Daniel R. Weinberger
- Andreas Meyer-Lindenberg
- Georg Northoff
- Joseph L. Price
In Experimental and Cognitive Psychology
- James J. Gross
- Kevin N. Ochsner
- A. Beck
- E. Gullone
- Andreas Meyer-Lindenberg
- K. Phan
- Mike Angstadt
- Pradeep J. Nathan
In Psychiatry and Mental health
- Daniel R. Weinberger
- Andreas Meyer-Lindenberg
- Joseph L. Price
- Robert J. DeRubeis
- Venkata S. Mattay
- Bhaskar S. Kolachana
- Georg Northoff
- A. Beck
Citations in the corpus
- The polyvagal perspective. (2007, background): “Consistent with the polyvagal theory, others have proposed direct neural pathways that integrate emotional activity with central structures that influence autonomic function (see Phillips et al., 2003a,b; Thayer and Lane, 2000; Craig, 2005).”
- A Review on the Computational Methods for Emotional State Estimation from the Human EEG (2013, background): “The ventral system, including ventral anterior cingulate gyrus and some ventral areas of prefrontal cortex (ventromedial prefrontal cortex and medial orbitofrontal cortex), is involved in the production of emotional states and the regulation of affective responses, whereas the dorsal system, includi”
- Amygdala-frontal connectivity during emotion regulation. (2007, background): “Given that a number of psychiatric disorders are associated with affective instability and emotion dysregulation (Koenigsberg et al., 2002; Phillips et al., 2003b), paradigms and analyses such as the one presented here should be extended to studies of patients with anxiety, mood and personality…”
- The evolution of the cognitive model of depression and its neurobiological correlates. (2008, background): “However, the findings need to be considered within a broader framework, including many brain regions implicated in depression (43, 44).”
- Are you always on my mind? A review of how face perception and attention interact. (2007, background): “…as autism, Williams syndrome, Huntington's disease, obsessive–compulsive disorder , social phobia, alcoholism, post-traumatic stress disorder, and schizophrenia, are characterized by impaired processing of facial identity and/or facial expression (Green & Phillips, 2004; Phillips et al., 2003b).”
- Imbalance between left and right dorsolateral prefrontal cortex in major depression is linked to negative emotional judgment: an fMRI study in severe major depressive disorder. (2008, background): “Emotional intensity rating scores ranged on a continuum from low (1) to high (9).”
- Observational fear learning involves affective pain system and Cav1.2 Ca2+ channels in ACC (2010, background): “Many aberrant social behaviors associated with psychiatric conditions, including various psychopathic or mental disorders (for example, post-traumatic stress disorders, schizophrenia, autism and dementia), feature impairment of recognition of the emotions and feelings of others and dysfunctions in t”
- Whole Brain Resting-State Analysis Reveals Decreased Functional Connectivity in Major Depression (2010, neutral): “An imbalanced functional integration of these subsystems may lead to a heightened response to negative information in ventral regions (bottom–up) on the one hand and a failure to regulate this response through dorsal regions (top–down) on the other (Phillips et al., 2003).”
- 5-HTTLPR polymorphism impacts human cingulate-amygdala interactions: a genetic susceptibility mechanism for depression (2005, background): “Covariance of gray matter structures Given the prior anatomical evidence of interconnection between amygdala and pAC”
- Cognitive therapy vs. medications for depression: Treatment outcomes and neural mechanisms (2008, background): “, and some accounts of brain abnormalities in patients with depression have emphasized the significance of neurochemical systems rather than brain region”
- Rapid and Longer-Term Antidepressant Effects of Repeated Ketamine Infusions in Treatment-Resistant Major Depression (2012, neutral): “Most individuals who respond to ketamine experience a relapse within several days or up to a week, although there is considerable variability in time to relapse following a single infusion (5, 12, 13).”
- Neuroimaging cognitive reappraisal in clinical populations to define neural targets for enhancing emotion regulation. A systematic review (2016, background): “This network includes lateral prefrontal regions (vlPFC, dlPFC) generally implicated in emotion regulation (Ochsner and Gross, 2005; Phillips et al., 2003), but extends beyond this core network to the dACC and inferior/superior parietal cortex, known to be involved in allocating resources during…”
- From inflammation to sickness and depression: when the immune system subjugates the brain (2008, result): “This would be consistent with the proposed implication of these brain areas in affective disorder”
- Psychobiology and molecular genetics of resilience (2009, neutral): “A neural model of emotion regulation consisting of ventral and dorsal systems has been described, with various patterns of abnormalities associated with a range of psychiatric disorder”
- New insights into symptoms and neurocircuit function of anorexia nervosa (2009, neutral): “Two neurocircuits that have been described based on imaging, neurophysiological and lesion studies 29,30 might be of particular relevance to understanding behaviour in AN.”
- Prefrontal-subcortical pathways mediating successful emotion regulation. (2008, neutral): “In the worst of circumstances, regulatory failures take a severe toll and contribute to the genesis and symptomatology of many psychiatric disorders (Davidson et al., 2000; Phillips et al., 2003).”