Age‐related intrinsic changes in human bone‐marrow‐derived mesenchymal stem cells and their differentiation to osteoblasts
By Shuanhu Zhou, J. Greenberger, M. Epperly, J. Goff, C. Adler, M. LeBoff, J. Glowacki. Aging Cell, 2008
751 citations recorded by the source corpus.
Subfields: Genetics, Oncology, Physiology
View Paper: https://www.semanticscholar.org/paper/e1dbaf0b4a2699c09510a629e36f421f96617216
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Citations in the corpus
- Mesenchymal stem cell-macrophage crosstalk and bone healing (2018, background): “overexpression of p53, p21, BAX or MYC genes, among other changes [72-75].”
- p38MAPK is a novel DNA damage response‐independent regulator of the senescence‐associated secretory phenotype (2011, background): “Thus, senescent cells, which increase with age in vivo (Dimri et al, 1995; Paradis et al, 2001; Jeyapalan et al, 2007; Zhou et al, 2008), may contribute to the low-level chronic inflammation that is a hallmark of aged mammalian tissues and many age-related diseases (Freund et al, 2010; Coppe et al,…”