Age-related inflammatory cytokines and disease.
By Helle Brüünsgaard, B. K. Pedersen. Immunology and allergy clinics of North America, 2003
625 citations recorded by the source corpus.
Subfields: Epidemiology, Immunology, Physiology
View Paper: https://www.semanticscholar.org/paper/d138e423c2ca6ba335c9e0c3e0312ed51d62726b
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Citations in the corpus
- Visceral Adiposity Index (2010, background): “V isceral obesity (1) is associated with increased adipocytokine production, proinflammatory activity (2), deterioration of insulin sensitivity (3), increased risk of developing diabetes, “high-triglyceride/low–HDL cholesterol dyslipidemia,” hypertension, atherosclerosis, and higher mortality rate (”
- Cellular senescence and the senescent secretory phenotype: therapeutic opportunities. (2013, positive): “Hallmarks of inflammation, including elevated IL-6, TNF-α , and immune cell chemokines, are associated with dementias (2), depression (3), atherosclerosis (4–8), cancers (9–11), diabetes (12– 14), and mortality (2, 15, 16).”
- Muscles and their myokines (2011, background): “Systemic low-level inflammation is defined as 2- to 4-fold elevations in circulating levels of pro-inflammatory and anti-inflammatory cytokines, naturally occurring cytokine antagonists, and acute-phase proteins, as well as minor increases in counts of neutrophils and natural killer cells (Bruunsgaa”
- Inflammatory mediators in the elderly. (2004, background): “Furthermore, TNF-a as well as IL-6 affect the coagulation system and metabolism of lipids, causing a procoagulant state and dyslipidaemia (reviewed in Bruunsgaard and Pedersen (2003)).”
- Muscle as an endocrine organ: focus on muscle-derived interleukin-6. (2008, background): “Systemic low-level inflammation is defined as two- to fourfold elevations in circulating levels of proinflammatory and anti-inflammatory cytokines, naturally occurring cytokine antagonists, and acute-phase proteins, as well as minor increases in counts of neutrophils and natural killer cells (45).”