Leukocyte-endothelial-cell interactions in leukocyte transmigration and the inflammatory response.

By Werner Müller. Trends in immunology, 2003

874 citations recorded by the source corpus.

Subfields: Immunology, Immunology and Allergy

View Paper: https://www.semanticscholar.org/paper/bac82f91405441c27c13be8da7bf5cb6971b97cc

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Citations in the corpus

Blood–brain Barrier: Structural Components and Function Under Physiologic and Pathologic Conditions (2006, background): “Increased Ca2+ flux leads to increase in isometric tension in endothelial cells.”
Pathophysiology of Atherosclerosis (2022, background): “In addition, some endothelial junction molecules actively mediate this type of migration [135,136].”
A transmigratory cup in leukocyte diapedesis both through individual vascular endothelial cells and between them (2004, background): “Second, models involving junctionally localized adhesion molecules are predicated on physiologic leukocyte diapedesis occurring at endothelial cell–cell junctions, i.e., the paracellular route of TEM (Bianchi et al., 1997; Muller, 2001, 2003; Aurrand-Lions et al., 2002).”
The role of adherens junctions and VE-cadherin in the control of vascular permeability (2008, background): “This function must be tightly regulated to maintain endothelial integrity and to prevent exposure of the subendothelial matrix of blood vessels, a highly thrombogenic event (Dejana, 2004; Muller, 2003; Vestweber, 2007).”
Getting to the site of inflammation: the leukocyte adhesion cascade updated (2007, background): “, into the post-adhesion events that strengthen leukocyte attachment to the endothelium, and into the molecules that are involved in leukocyte transendothelial migratio”
The blood-brain barrier in Alzheimer's disease (2017, positive): “Interendothelial adhesive proteins control endothelial cell – cell interactions, but some may also participate in leukocyte transendothelial migration via homophilic or heterophilic interactions (Muller, 2003; Tietz and Engelhardt, 2015).”
Adhesion mechanisms regulating the migration of monocytes (2004, background): “PECAM1 has been implicated as a crucial mediator of monocyte and neutrophil transendothelial migratio”
Immune cell migration in inflammation: present and future therapeutic targets (2005, background): “Diapedesis in distinct inflammatory settings also indicates sequential occupancy by the migrating leukocytes of constitutively expressed junctional endothelial ligands such as PECAM-1, CD99 and JAM”
Mechanisms of Leukocyte Transendothelial Migration (2011, background): “Because leukocytes cannot swim, they are recruited locally at the site of inflammation in a series of adhesive steps that allow them to attach to the vessel wall, locomote along the wall to the endothelial borders, traverse the endothelium and the subendothelial basement membrane, and migrate throug”
A Review of Cell Adhesion Studies for Biomedical and Biological Applications (2015, background): “The post-adhesion events strengthen cell attachment to the endothelium, and into the molecules that are involved in cells’ transendothelial migration [45,46].”
Endothelial cell–cell junctions: happy together (2004, background): “This process is usually very rapid and is followed by an equally rapid reassembly of junctions, which prevents increased permeabilit”