Endothelial cell–cell junctions: happy together

By E. Dejana. Nature reviews. Molecular cell biology, 2004

1,248 citations recorded by the source corpus.

Subfields: Cell Biology, Immunology and Allergy, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/66a5eb249f02af9ef3fca249a25b821b8f1232f9

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cell Biology

1. F. Orsenigo
2. M. Lampugnani
3. F. van Roy
4. G. Berx
5. S. Nourshargh
6. E. Dejana
7. Peter C. Searson
8. P. Kubes

In Immunology and Allergy

1. F. van Roy
2. G. Berx
3. S. Nourshargh
4. F. Orsenigo
5. E. Dejana
6. P. Kubes
7. F. Tacke
8. L. Hammerich

In Molecular Biology

1. F. Orsenigo
2. M. Lampugnani
3. G. Berx
4. F. van Roy
5. S. Nourshargh
6. M. Potente
7. E. Dejana
8. Klaus Ley

Citations in the corpus

• The blood-brain barrier: an engineering perspective (2013, background): “Endothelial adherens junctions are formed by the extracellular domains of vascular endothelial cadherin (VE-cadherin) and are linked to the actin cytoskeleton inside the cell via proteins such as α-catenin, β-catenin, and vinculin (Bazzoni and Dejana, 2004; Dejana, 2004).”
• VEGF controls endothelial-cell permeability by promoting the β-arrestin-dependent endocytosis of VE-cadherin (2006, neutral): “, an endothelial specific cell–cell adhesion molecul”
• The role of adherens junctions and VE-cadherin in the control of vascular permeability (2008, background): “(Wallez et al., 2006) CSK Tyrosine kinase (substrate: SRC) Direct (phosphoY685) (Baumeister et al., 2005) PYK2 Tyrosine kinase (substrate: VE-cadherin?) n.d. (Allingham et al., 2007) PAK Ser-Thr kinase (substrate: VE-cadherin S665) n.d. (Gavard and Gutkind, 2006) SHP2 Tyrosine phosphatase…”
• Endothelial adherens junctions control tight junctions by VE-cadherin-mediated upregulation of claudin-5 (2008, background): “This unique characteristic is largely exerted by the coordinated opening and closure of cell–cell junction”
• TGFbeta primes breast tumors for lung metastasis seeding through angiopoietin-like 4. (2008, neutral): “Staining with antibodies against the tight junction component zonula occludens 1 (ZO-1), against the adherens junction component βcatenin, or staining of the actin cytoskeleton with phalloidin (Dejana, 2004), revealed that the monolayer integrity was dramatically perturbed by Angptl4 (Figure 6A and ”
• Vascular permeability, vascular hyperpermeability and angiogenesis (2008, neutral): “[61–63].”
• Getting to the site of inflammation: the leukocyte adhesion cascade updated (2007, background): “For example, nectins JAM-A and PECAM1, through interactions with common cytoplasmic partners (such as catenins), are connected with the cadherin syste”