Proinflammatory cytokines, aging, and age-related diseases.
By M. Michaud, L. Balardy, G. Moulis, C. Gaudin, C. Peyrot, B. Vellas, M. Cesari, F. Nourhashemi. Journal of the American Medical Directors Association, 2013
1,026 citations recorded by the source corpus.
Subfields: Epidemiology, Neurology, Physiology
View Paper: https://www.semanticscholar.org/paper/2f74039d5cf6de15424d11a38b0705b7338fb8d2
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Citations in the corpus
- JAK inhibition alleviates the cellular senescence-associated secretory phenotype and frailty in old age (2015, background): “Inflammation also is associated with frailty, a geriatric syndrome characterized by decreased strength and incapacity to respond to stress (2).”
- Antioxidant and Oxidative Stress: A Mutual Interplay in Age-Related Diseases (2018, background): “COX-derived reactive species and transcriptional activity of interleukin-1beta (IL-1β), interleukin-6 (IL-6), tumor necrosis factor-α (TNF-α), cyclooxygenase-2 (COX-2), and inducible nitric oxide synthase (iNOS) are increased during aging (Michaud et al., 2013; Zhang and Jiang, 2015; PuzianowskaKuzn”