Oxidative stress and gene regulation.
By R. G. Allen, M. Tresini. Free Radical Biology & Medicine, 2000
1,312 citations recorded by the source corpus.
Subfields: Molecular Biology, Nutrition and Dietetics
View Paper: https://www.semanticscholar.org/paper/9502ca87d679977baed15328f63c6a37d708ddff
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Molecular Biology
- S. Neill
- J. Hancock
- Radhika Desikan
- J. Nordberg
- Kelsey H. Fisher-Wellman
- Constantinos Fiotakis
- Elias S. J. Arnér
- M. Jackson
In Nutrition and Dietetics
Citations in the corpus
- Exercise-Induced Oxidative Stress: Cellular Mechanisms and Impact on Muscle Force Production (2008, background): “While it is also generally assumed that these effects are modulated by ROS-induced oxidation, antioxidants can also stimulate some specific gene expression pathways (12).”
- Glucocorticoid-Mediated Phenotypes in Vertebrates : Multilevel Variation and Evolution (2016, background): “However, other mechanisms may contribute to this phenomenon, including age-dependent changes in hippocampal GRs (Stein-Behrens & Sapolsky, 1992).”
- Comprehending crystalline β-carotene accumulation by comparing engineered cell models and the natural carotenoid-rich system of citrus (2011, neutral): “Oxidation-related genes have been extensively studied under drought stress at the molecular level (Allen and Tresini, 2000).”
- Oxidative Stress and Antioxidant Responses in Young Leaves of Mulberry Plants Grown Under Nitrogen, Phosphorus or Potassium Deficiency (2007, background): “ROS are also suggested to serve as messengers in gene regulatory and signal transduction pathways (Allen and Tresini 2000; Shin and Schachtman 2004) and have been implicated in the modulation of gene expression by redox mediated transcriptional factors (Dinesh-Kumar et al.”
- Exercise is the real polypill. (2013, background): “Other pathways are MAPK, PI3K/Akt, or p53 activation (11, 203, 361).”
- Regulation of the Arabidopsis transcriptome by oxidative stress. (2001, background): “Although ROS were originally considered to be detrimental to cells, it is now widely recognized that redox regulation involving ROS is a key factor modulating cellular activities (Allen and Tresini, 2000; Dat et al., 2000).”
- Novel mechanisms of natural antioxidant compounds in biological systems: involvement of glutathione and glutathione-related enzymes. (2005, background): “[42] Allen RG, Tresini M.”
- 8-hydroxy-2′ -deoxyguanosine (8-OHdG): A Critical Biomarker of Oxidative Stress and Carcinogenesis (2009, background): “Also, epigenetic effects by ROS and oxidative stress, such as various types of signal transduction mechanisms, lead to apoptosis, modulate cell proliferation, and immune response, thus complicating the estimation of risk ( 71 , 72 , 73 ).”
- Ionizing radiation-induced metabolic oxidative stress and prolonged cell injury (2011, background): “At low levels, ROS participate in signaling pathways that maintain normal cellular functions by regulating the expression of specific genes [100; 101; 102; 103], modulating ion channel activities [104], and mimicking or affecting intermediates (e.”
- Anthocyanins: natural colorants with health-promoting properties. (2010, background): “However, if ROS are overly produced, they can elicit cellular damage, leading to degenerative diseases such as inflammation, cardiovascular disease, cancer, and aging (Allen & Tresini 2000).”