Can we conquer pain?
By J. Scholz, Clifford J. Woolf. Nature Neuroscience, 2002
883 citations recorded by the source corpus.
Subfields: Cellular and Molecular Neuroscience, Cognitive Neuroscience, Physiology
View Paper: https://www.semanticscholar.org/paper/ca36194e060226a5060e8eaa4c835d4574891741
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In Cellular and Molecular Neuroscience
In Cognitive Neuroscience
In Physiology
Citations in the corpus
- The effect of pain on cognitive function: a review of clinical and preclinical research. (2011, background): “NMDA receptors are also implicated in central sensitization and wind-up (Scholz and Woolf, 2002).”
- Pain: Moving from Symptom Control toward Mechanism-Specific Pharmacologic Management (2004, background): “Multiple mechanisms that can produce pain have been identified; they include nociception, peripheral sensitization, phenotypic switches, central sensitization, ectopic excitability, structural reorganization, and decreased inhibition (1, 2, 14, 15).”
- Predictors of Postoperative Pain and Analgesic Consumption: A Qualitative Systematic Review (2009, background): “Intense influx of pain signal from tissue trauma after surgery can lead to enhancement of excitability and responsiveness of dorsal horn neurons to pain transmission.(84) Their excitability can be further sustained by transcriptional changes, such as induction of genes cyclooxygenase-2 (COX-2) inhib”