[Obesity and insulin resistance].
By Y. Tamori, M. Kasuga. Nihon rinsho. Japanese journal of clinical medicine, 2009
1,489 citations recorded by the source corpus.
View Paper: https://www.semanticscholar.org/paper/798efcbb54c9a7580c706027a2e01399c7a9ae30
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Citations in the corpus
- Hypoxia is a potential risk factor for chronic inflammation and adiponectin reduction in adipose tissue of ob/ob and dietary obese mice. (2007, background): “IN OBESITY, chronic inflammation and reduced adiponectin in the white adipose tissue (WAT) contribute to pathogenesis of insulin resistance, which links obesity to many complications, such as type 2 diabetes and cardiovascular diseases (23, 24, 30).”
- Obesity and Its Metabolic Complications: The Role of Adipokines and the Relationship between Obesity, Inflammation, Insulin Resistance, Dyslipidemia and Nonalcoholic Fatty Liver Disease (2014, background): “The free fatty acids from enlarged adipose tissue are then taken up by the hepatocytes, which lead to reduced hepatic insulin clearance with a further increase in circulating insulin levels [53].”
- Macrophage-mediated inflammation in metabolic disease (2011, background): “Obesity-induced insulin resistance The resistance of metabolic tissues — such as adipose tissue, liver and muscle — to the anabolic actions of insulin is termed insulin resistance, a characteristic feature of obesity-induced metabolic dysfunctio”
- Obesity and colon and rectal cancer risk: a meta-analysis of prospective studies. (2007, background): “Obesity, especially abdominal obesity, is related to insulin resistance and hyperinsulinemia (5, 6).”
- Cardiac remodeling in obesity. (2008, background): “The development of insulin resistance and type 2 diabetes in obese individuals (163, 219), the significant contribution of diabetes to cardiovascular disease (234, 255, 352), and potential mechanisms by which diabetes may lead to cardiac dysfunction (31) have been extensively reviewed.”
- The metabolic syndrome. (2008, background): “In the setting of insulin resistance and expanded adipose tissue triglyceride stores, the process of FFA mobilization (lipolysis) from stored adipose tissue triglyceride is accelerated (7, 127).”
- Bifurcation of insulin signaling pathway in rat liver: mTORC1 required for stimulation of lipogenesis, but not inhibition of gluconeogenesis (2010, background): “O frequently leads to insulin-resistance that, in turn, produces Type 2 diabetes (1, 2).”
- Trends in insulin resistance: insights into mechanisms and therapeutic strategy (2022, methodology): “THE DIAGNOSIS AND THERAPEUTIC STRATEGY OF IR Diagnosis methods of IR As we all know, IR is related to several metabolic abnormalities including obesity, glucose tolerance, dyslipidemia, type 2 diabetes and other metabolic syndrome.(429,430) Several methods are used to measure blood insulin levels th”
- Selective increases of bifidobacteria in gut microflora improve high-fat-diet-induced diabetes in mice through a mechanism associated with endotoxaemia (2007, background): “Evidence suggests that obesity and metabolic disorders (type 2 diabetes and insulin resistance) are tightly linked to inflammation [1, 2].”
- Endothelial dysfunction in diabetes mellitus (2007, unknown): “In contrast to patients with type 1 diabetes, endothelial dysfunction can also occur in patients with type 2 diabetes even when the patients have normal urinary albumin excretion.”