[Mitochondria and apoptosis].

By Y. Li, Z. Li. Zhonghua yu fang yi xue za zhi [Chinese journal of preventive medicine], 2000

4,378 citations recorded by the source corpus.

View Paper: https://www.semanticscholar.org/paper/78169064f56af88c851bab6d13b2e4ca53f35eb6

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Citations in the corpus

PUMA, a novel proapoptotic gene, is induced by p53. (2001, background): “The second apoptotic pathway is associated with release of cytochrome c from mitochrondria and activation of Apaf-1Summary (Green and Reed, 1998).”
Mitochondrial reactive oxygen species in cell death signaling. (2002, background): “[4] B.”
The Hallmarks of Cancer Review (2000, background): “Many of the signals that elicit apoptosis converge on the mitochondria, which respond to proapoptotic signals by releasing cytochrome C, a potent catalyst of apoptosis (Green and Reed, 1998).”
Inhibition of early apoptotic events by Akt/PKB is dependent on the first committed step of glycolysis and mitochondrial hexokinase. (2001, background): “Mitochondria play a major role in this process through the release of cytochrome c and other proapoptotic proteins that normally reside in the intermembrane space between the inner and outer mitochondrial membranes (for review, see Gross et al. 1999; Desagher and Martinou 2000).”
Mitochondrial dysfunction as a cause of optic neuropathies. (2004, background): “…stress, mitochondrial inner membrane potential, and calcium fluxes are all intimately involved in controlling the opening of the mitochondrial permeability transition pore (MPTP) through which the pro-apoptotic factors are released into the cytosol (Zoratti and Szabo, 1995; Green and Reed, 1998).”
Calcium, ATP, and ROS: a mitochondrial love-hate triangle. (2004, background): “Studies on isolated mitochondria with other ROS probes have provided more information on this scenario, showing that a complex interplay exists between PT pore opening and ROS generation in response to Ca(2) (29, 61, 102, 204).”
BNIP3 and Genetic Control of Necrosis-Like Cell Death through the Mitochondrial Permeability Transition Pore (2000, background): “One of the key events in apoptosis is the release of cytochrome c, which functions with dATP as a cofactor for Apaf-1 activation of the caspase cascade (14).”
The possibility of neurotoxicity in the hippocampus in major depression: a primer on neuron death. (2000, background): “More sensitive means to detect apoptosis, developed in the early 1990s, produced the surprising observation that glutamatergic insults trigger apoptosis in a subset of moribund neurons (Green and Reed 1998; Roy and Sapolsky 1999).”
Essential role of the mitochondrial apoptosis-inducing factor in programmed cell death (2001, methodology): “aif–/Y (1) aifneo/Y (1) aifneo/Y (2) aif–/Y (2) aif–/Y (3)”
The role and regulation of programmed cell death in plant–pathogen interactions (2004, background): “In animals, apoptosis often involves proteases called caspases (Green and Reed, 1998).”
The Biology of Forgetting – A Perspective (2017, background): “The complex process of apoptosis, which occurs via several different biochemical pathways, each involving numerous molecular steps, actively removes the extra cells (Green and Reed, 1998).”
Apoptosis and Molecular Targeting Therapy in Cancer (2014, background): “Many tumors refract to the classic apoptosis-inducing cancer therapies which provoke intrinsic pathway-based apoptosis due to development of defects in that pathway under treatment stress [233].”
Proteases for Cell Suicide: Functions and Regulation of Caspases (2000, background): “However, cytochrome c release and caspase activation can occur before any detectable changes in mitochondrial morphology (72).”