International Union of Pharmacology. LXI. Peroxisome Proliferator-Activated Receptors
By L. Michalik, Johan Auwerx, J. Berger, V. Chatterjee, Christopher K. Glass, F. Gonzalez, P. Grimaldi, Takashi Kadowaki, Mitchell A. Lazar, S. O’Rahilly and 6 more. Pharmacological Reviews, 2006
1,003 citations recorded by the source corpus.
Subfields: Molecular Biology, Pharmacology, Physiology
View Paper: https://www.semanticscholar.org/paper/23952f6b64ae102e2d6ce0fdcb80b46efc4150a7
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Citations in the corpus
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (2012, neutral): “proliferator-activated receptors.(772) Degradation of excess organelles is induced by drug withdrawal, although starvation without prior proliferation can also be used.”
- Natural product agonists of peroxisome proliferator-activated receptor gamma (PPARγ): a review (2014, background): “fatty acids, prostanoids) [19,26,144–151] and synthetic [13,151–153] ligands of the receptor have been reviewed in numerous previous articles and therefore will not be discussed here.”
- International Union of Basic and Clinical Pharmacology. LXXIX. Cannabinoid Receptors and Their Ligands: Beyond CB1 and CB2 (2010, background): “Peroxisome proliferator-activated receptors (PPARs) are ligand-activated transcription factors that constitute part of the nuclear receptor family (Michalik et al., 2006).”
- The role of Peroxisome Proliferator-Activated Receptors (PPAR) in immune responses (2020, background): “Each PPAR member has a different ligand preference that could be explained by differences in the size or the lipophilicity of their binding pockets [8].”
- Towards Fish Lipid Nutrigenomics: Current State and Prospects for Fin-Fish Aquaculture (2008, background): “This has led to the view that PPARs are general fatty acid sensors responding to increased cellular fatty acid levels or their metabolites, which arise as a result of changes in nutritional status and energy metabolism (Michalik et al., 2006).”