Mitogen-activated protein (MAP) kinase pathways: regulation and physiological functions.

By G. Pearson, Fred L Robinson, T. Gibson, Bing-e Xu, M. Karandikar, K. Berman, M. Cobb. Endocrine reviews, 2001

4,259 citations recorded by the source corpus.

View Paper: https://www.semanticscholar.org/paper/6158e791be5727e7ee63f3259f4eeadf88de1a2a

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Citations in the corpus

Neurotrophin-regulated signalling pathways (2006, background): “Data suggest that secretion of proneurotrophins is increased following brain injury or degeneration, and binding of these proteins to p75NTR may increase neuronal loss in these injury and disease models (Fahnestock et al. 2001; Harrington et al. 2004; Pedraza et al. 2005).”
Mitogen-activated protein kinase signaling in the heart: angels versus demons in a heart-breaking tale. (2010, background): “These biological events are the culmination of signal transduction and regulation by primarily four MAPK subfamilies including extracellular signal-regulated kinases (ERK1/2), c-Jun NH2-terminal kinases (JNK1, -2 and -3), p38 kinase ( , , , ), and big MAPK (BMK or ERK5) (185, 318, 329).”
Inflammatory responses and inflammation-associated diseases in organs (2015, background): “com/oncotarget present in the cytoplasm or nucleus, which initiates the inflammatory response [32, 36] (Figure 3).”
Quantitative Phosphoproteomics of Early Elicitor Signaling in Arabidopsis*S (2007, background): “MAP kinases phosphorylate serine or threonine residues followed by a proline in both plant and animal systems (53, 54).”
The role of beta-arrestins in the termination and transduction of G-protein-coupled receptor signals. (2002, background): “In addition to directly phosphorylating nuclear transcription factors, ERK1/2 phosphorylates numerous plasma membrane, cytoplasmic and cytoskeletal substrates (Pearson et al., 2001).”
p38 MAP kinases: key signalling molecules as therapeutic targets for inflammatory diseases (2003, background): “So far, three distinct MAPK pathways have been described in mammalian cells, which seem to be conserved from yeast to ma”
MAPK cascade signalling and synaptic plasticity (2004, background): “jpg" NDATA ITEM> ]> Several MAPK cascades have been characterized in mammalian cells (reviewed extensively by Cobb and colleague”
Nuclear retinoid receptors and the transcription of retinoid-target genes. (2004, neutral): “1) (Morgan, 1997; Davis, 2000; Pearson et al., 2001).”
The interplay between the glucocorticoid receptor and nuclear factor-kappaB or activator protein-1: molecular mechanisms for gene repression. (2003, background): “Many proinflammatory cytokines, aberrantly up-regulated in chronic inflammatory diseases, require the concerted activation of NF- B and AP-1, which are positively controlled by MAPK cascades (708, 709).”