Requirement of interleukin-17A for systemic anti-Candida albicans host defense in mice.

By Weitao Huang, L. Na, P. Fidel, P. Schwarzenberger. Journal of Infectious Diseases, 2004

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IL-17 and Th17 Cells. (2009, background): “Requirement of interleukin-17A for systemic anti-Candida albicans host defense in mice.”
The IL-23/Th17 axis in the immunopathogenesis of psoriasis. (2009, background): “IL-17R-deficient mice are highly susceptible to infection by the Gram-negative bacterium, Klebsiella pneumoniae, and the fungus, Candida albicans (Ye et al., 2001; Huang et al., 2004).”
Interleukin-17-producing gammadelta T cells selectively expand in response to pathogen products and environmental signals. (2009, background): “…in peripheral tissues for pathogen clearance (Forlow et al., 2001; Ye et al., 2001b) and host defense against Klebsiella or Candida infections (Huang et al., 2004; Ye et al., 2001a), and it can also protect mucosal barrier functions by stimulating tight junction formation and mucin secretion…”
IL-17-Mediated Regulation of Innate and Acquired Immune Response against Pulmonary Mycobacterium bovis Bacille Calmette-Guérin Infection1 (2007, background): “against Klebsiella or Candida infections (19, 20), rheumatoid ar-”