Neuron number in the entorhinal cortex and CA1 in preclinical Alzheimer disease.

By Joseph L. Price, Andy I. Ko, M. Wade, Sarah K. Tsou, D. McKeel, J. Morris. Archives of Neurology, 2001

613 citations recorded by the source corpus.

Subfields: Cognitive Neuroscience, Physiology, Psychiatry and Mental health

View Paper: https://www.semanticscholar.org/paper/4e97d3060d723cab36cb2f2f5ec81498e8ee56fd

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Citations in the corpus

Alzheimer’s Disease: The Challenge of the Second Century (2011, background): “By the time very mild dementia/MCI is present, in addition to substantial densities of plaques and tangles, there is also significant neuronal loss in certain brain regions (9, 211).”
Automated detection of brain atrophy patterns based on MRI for the prediction of Alzheimer's disease (2010, result): “…pattern of atrophy detected in the current study agrees with ﬁ ndings of a range of previous independent MRI based (Meguro et al., 2001; Scahill et al., 2002) and neuropathological studies showing AD-typical predilection sites of pathological changes (Braak et al., 1997; Price et al., 1991, 2001).”
Neuropathological alterations in Alzheimer disease. (2011, background): “1996), and they do not seem to have significant neuronal loss, not even in vulnerable regions such as the entorhinal cortex and the hippocampus (Price et al. 2001; West et al. 2004).”
Inverse relation between in vivo amyloid imaging load and cerebrospinal fluid Aβ42 in humans (2006, background): “Thus, our data suggest that a specific plaqueassociated alteration in A 42 metabolism is involved in the PIB/CSF A 42 association, because the nondemented subjects in our cohort are unlikely to have appreciable neuronal loss given our prior findings.(20) Regardless of the underlying mechanism(s), th”
Age-related myelin breakdown: a developmental model of cognitive decline and Alzheimer's disease. (2004, background): “[191] Price JL, Ko AI, Wade MJ, Tsou SK, McKeel DW, Morris JC.”
Memory and executive function in aging and AD: multiple factors that cause decline and reserve factors that compensate. (2004, background): “…stages of the disease who, because of high baseline lobe early in the disease and spreads outward to associfunctioning and other mitigating factors, retain averageation cortex over time (Braak and Braak, 1991; 1997; global cognition during the initial progression of the dis-Price et al., 2001).”
Neuroprotective effects of brain-derived neurotrophic factor in rodent and primate models of Alzheimer’s disease (2009, background): “1912" language="eng" relation="no"> Profound neuronal dysfunction in the entorhinal cortex contributes to early loss of short-term memory in Alzheimer's diseas”
Differential aging of the brain: patterns, cognitive correlates and modifiers. (2006, neutral): “Furthermore, the EC is affected almost exclusively in those who showed significant ante-mortem cognitive decline (Kordower et al., 2001; Price et al., 2001).”
Western Diet Consumption and Cognitive Impairment: Links to Hippocampal Dysfunction and Obesity (2010, background): “In contrast, the hippocampus is the site of structural abnormalities associated with early stages of AD and other cognitive dementias [8,9].”