J. Fletcher

Molecular Biology · Molecules and Cells · 2005–2010

A researcher in the Science corpus: 5 papers; trusts 1 researcher and is trusted by 2 researchers here, as inferred from the citation graph.

Subfields

Whom J. Fletcher trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Jerry M. Adams

Who trusts J. Fletcher overall

  1. Balázs Győrffy
  2. Jerry M. Adams

Papers from J. Fletcher's perspective

Ordered by how strongly the researchers J. Fletcher trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Noxa, a BH3-only member of the Bcl-2 family and candidate mediator of p53-induced apoptosis. (Science)
  2. The combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues. (Molecules and Cells)
  3. Identification of DIABLO, a mammalian protein that promotes apoptosis by binding to and antagonizing IAP proteins. (Cell)
  4. Elimination of Mcl-1 is required for the initiation of apoptosis following ultraviolet irradiation. (Genes & Development)
  5. Proapoptotic BAX and BAK: A Requisite Gateway to Mitochondrial Dysfunction and Death (Science)
  6. VDAC2 Inhibits BAK Activation and Mitochondrial Apoptosis (Science)
  7. Bax Is Present as a High Molecular Weight Oligomer/Complex in the Mitochondrial Membrane of Apoptotic Cells* (Journal of Biological Chemistry)
  8. Bax and Bak can localize to the endoplasmic reticulum to initiate apoptosis (Journal of Cell Biology)
  9. Obligate Role of Anti-Apoptotic MCL-1 in the Survival of Hematopoietic Stem Cells (Science)
  10. The Pathophysiology of Mitochondrial Cell Death (Science)

Their papers