K. Stanhope
Endocrinology, Diabetes and Metabolism · 2009–2015
A researcher in the Science corpus: 2 papers; trusts 7 researchers and is trusted by 1 researcher here, as inferred from the citation graph.
Subfields
- Endocrinology, Diabetes and Metabolism — 24 citations into it; #557 of 623 by trust here, #314 by citations
- Physiology — 18 citations into it; #1,966 of 2,276 by trust here, #1,242 by citations
- Epidemiology — 11 citations into it; #1,893 of 2,148 by trust here, #1,062 by citations
- Molecular Biology — 7 citations into it
Whom K. Stanhope trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts K. Stanhope overall
Papers from K. Stanhope's perspective
Ordered by how strongly the researchers K. Stanhope trusts endorse each paper. Same corpus, a different order for every researcher.
- Lipid-induced insulin resistance in human muscle is associated with changes in diacylglycerol, protein kinase C, and IkappaB-alpha. (Diabetes)
- Fat accumulation in the liver is associated with defects in insulin suppression of glucose production and serum free fatty acids independent of obesity in normal men. (Journal of Clinical Endocrinology and Metabolism)
- ATVB In Focus Metabolic Syndrome and Atherosclerosis
- Relation between consumption of sugar-sweetened drinks and childhood obesity: a prospective, observational analysis. (The Lancet)
- Sucrose compared with artificial sweeteners: different effects on ad libitum food intake and body weight after 10 wk of supplementation in overweight subjects. (American Journal of Clinical Nutrition)
- Follow-up report on the diagnosis of diabetes mellitus. (Diabetes Care)
- Molecular Mechanisms of Insulin Resistance in Humans and Their Potential Links With Mitochondrial Dysfunction (Diabetes)
- Overproduction of large VLDL particles is driven by increased liver fat content in man (Diabetologia)
- Regulation of the ABC kinases by phosphorylation: protein kinase C as a paradigm. (Biochemical Journal)
- Lipoprotein lipase: structure, function, regulation, and role in disease (Journal of molecular medicine)