E. Jo

Molecular Biology · 2009–2021

A researcher in the Science corpus: 5 papers; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.

Subfields

Whom E. Jo trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Fabio Martinon
  2. Rongbin Zhou

Papers from E. Jo's perspective

Ordered by how strongly the researchers E. Jo trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Autophagy proteins regulate innate immune response by inhibiting NALP3 inflammasome-mediated mitochondrial DNA release (Nature Immunology)
  2. Fatty acid-induced NLRP3-PYCARD inflammasome activation interferes with insulin signaling (Nature Immunology)
  3. Oxidized mitochondrial DNA activates the NLRP3 inflammasome during apoptosis. (Immunity)
  4. Type 2 diabetes as an inflammatory disease (Nature reviews. Immunology)
  5. Mitochondrial reactive oxygen species promote production of proinflammatory cytokines and are elevated in TNFR1-associated periodic syndrome (TRAPS) (Journal of Experimental Medicine)
  6. Intracellular NOD-like receptors in host defense and disease. (Immunity)
  7. Differential requirement for the activation of the inflammasome for processing and release of IL-1beta in monocytes and macrophages. (Blood)
  8. Critical role for calcium mobilization in activation of the NLRP3 inflammasome (Proceedings of the National Academy of Sciences of the United States of America)
  9. K+ efflux is the Common Trigger of NLRP3 inflammasome Activation by Bacterial Toxins and Particulate Matter (Immunity)
  10. The calcium-sensing receptor regulates the NLRP3 inflammasome through Ca2+ and cAMP (Nature)

Their papers