C. X. Chen
Epidemiology · 2014
A researcher in the Science corpus: 1 paper; trusts 4 researchers and is trusted by 1 researcher here, as inferred from the citation graph.
Subfields
- Epidemiology — 21 citations into it; #1,676 of 2,148 by trust here, #1,657 by citations
- Neurology — 17 citations into it; #1,126 of 1,455 by trust here, #1,146 by citations
- Molecular Biology — 12 citations into it; #15,110 of 17,773 by trust here, #14,509 by citations
Whom C. X. Chen trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts C. X. Chen overall
Papers from C. X. Chen's perspective
Ordered by how strongly the researchers C. X. Chen trusts endorse each paper. Same corpus, a different order for every researcher.
- Parkin is recruited selectively to impaired mitochondria and promotes their autophagy (Journal of Cell Biology)
- PINK1 Is Selectively Stabilized on Impaired Mitochondria to Activate Parkin (PLoS Biology)
- PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1 (Nature Cell Biology)
- PINK1-dependent recruitment of Parkin to mitochondria in mitophagy (Proceedings of the National Academy of Sciences of the United States of America)
- Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin (Nature)
- Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin (Nature)
- Mitochondrial pathology and apoptotic muscle degeneration in Drosophila parkin mutants (Proceedings of the National Academy of Sciences of the United States of America)
- Fission and selective fusion govern mitochondrial segregation and elimination by autophagy (EMBO Journal)
- Drosophila Parkin requires PINK1 for mitochondrial translocation and ubiquitinates Mitofusin (Proceedings of the National Academy of Sciences of the United States of America)
- PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy (Journal of Cell Biology)