Wei-Chung Chiang
Epidemiology · 2016
A researcher in the Science corpus: 1 paper; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Epidemiology — 8 citations into it
Whom Wei-Chung Chiang trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Papers from Wei-Chung Chiang's perspective
Ordered by how strongly the researchers Wei-Chung Chiang trusts endorse each paper. Same corpus, a different order for every researcher.
- Broad activation of the ubiquitin–proteasome system by Parkin is critical for mitophagy (Human Molecular Genetics)
- To be or not to be? How selective autophagy and cell death govern cell fate. (Cell)
- Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin (Nature)
- Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin (Nature)
- Fission and selective fusion govern mitochondrial segregation and elimination by autophagy (EMBO Journal)
- PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1 (Nature Cell Biology)
- Parkin is recruited selectively to impaired mitochondria and promotes their autophagy (Journal of Cell Biology)
- A role for ubiquitin in selective autophagy. (Molecules and Cells)
- PINK1-dependent recruitment of Parkin to mitochondria in mitophagy (Proceedings of the National Academy of Sciences of the United States of America)
- Drosophila Parkin requires PINK1 for mitochondrial translocation and ubiquitinates Mitofusin (Proceedings of the National Academy of Sciences of the United States of America)
Their papers
- Prohibitin 2 Is an Inner Mitochondrial Membrane Mitophagy Receptor (2016) — 761 citations