Arturo V. Orjalo
Physiology · 2010–2015
A researcher in the Science corpus: 2 papers; trusts 2 researchers and is trusted by 1 researcher here, as inferred from the citation graph.
Subfields
- Physiology — 39 citations into it; #1,849 of 2,276 by trust here, #956 by citations
- Molecular Biology — 34 citations into it
Whom Arturo V. Orjalo trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts Arturo V. Orjalo overall
Papers from Arturo V. Orjalo's perspective
Ordered by how strongly the researchers Arturo V. Orjalo trusts endorse each paper. Same corpus, a different order for every researcher.
- Chemokine signaling via the CXCR2 receptor reinforces senescence. (Cell)
- Senescence of activated stellate cells limits liver fibrosis (Cell)
- Persistent DNA damage signaling triggers senescence-associated inflammatory cytokine secretion (Nature Cell Biology)
- Living on a break: cellular senescence as a DNA-damage response (Nature Reviews. Cancer)
- The Senescence-Associated Secretory Phenotype: The Dark Side of Tumor Suppression (Annual Review of Pathology)
- Senescence-messaging secretome: SMS-ing cellular stress (Nature Reviews. Cancer)
- Inflammaging and anti-inflammaging: a systemic perspective on aging and longevity emerged from studies in humans. (Mechanisms of Ageing and Development)
- Senescent cells, tumor suppression, and organismal aging: good citizens, bad neighbors. (Cell)
- Cellular senescence: when bad things happen to good cells (Nature reviews. Molecular cell biology)
- Interleukin-6 in aging and chronic disease: a magnificent pathway. (The journals of gerontology. Series A, Biological sciences and medical sciences)
Their papers
- MTOR regulates the pro-tumorigenic senescence-associated secretory phenotype by promoting IL1A translation (2015) — 1,061 citations
- Inflammatory Networks during Cellular Senescence: Causes and Consequences (2010) — 1,311 citations