Jon S. Cardinal
Clinical Biochemistry · 2008
A researcher in the Science corpus: 1 paper; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Clinical Biochemistry — 15 citations into it
Whom Jon S. Cardinal trusts
Their strongest outbound trust, strongest first, inferred from their citations.
- M. Bianchi (in Clinical Biochemistry)
Papers from Jon S. Cardinal's perspective
Ordered by how strongly the researchers Jon S. Cardinal trusts endorse each paper. Same corpus, a different order for every researcher.
- Reversing established sepsis with antagonists of endogenous high-mobility group box 1 (Proceedings of the National Academy of Sciences of the United States of America)
- Release of chromatin protein HMGB1 by necrotic cells triggers inflammation (Nature)
- The nuclear factor HMGB1 mediates hepatic injury after murine liver ischemia-reperfusion (Journal of Experimental Medicine)
- Involvement of Toll-like Receptors 2 and 4 in Cellular Activation by High Mobility Group Box 1 Protein* (Journal of Biological Chemistry)
- DAMPs, PAMPs and alarmins: all we need to know about danger (Journal of Leukocyte Biology)
- High-mobility group box 1 protein (HMGB1): nuclear weapon in the immune arsenal (Nature reviews. Immunology)
- High Mobility Group 1 Protein (Hmg-1) Stimulates Proinflammatory Cytokine Synthesis in Human Monocytes (Journal of Experimental Medicine)
- Alarmins: chemotactic activators of immune responses. (Current Opinion in Immunology)
- Toll-like receptor 9–dependent activation by DNA-containing immune complexes is mediated by HMGB1 and RAGE (Nature Immunology)
- Inflammation-promoting activity of HMGB1 on human microvascular endothelial cells. (Blood)
Their papers
- HMGB1: Endogenous Danger Signaling (2008) — 788 citations