Inflammation-promoting activity of HMGB1 on human microvascular endothelial cells.

By Carmen Fiuza, M. Bustin, Shefali Talwar, M. Tropea, E. Gerstenberger, J. Shelhamer, A. Suffredini. Blood, 2003

771 citations recorded by the source corpus.

Subfields: Clinical Biochemistry, Epidemiology, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/86325ac3fc44552dc3b0956e79e0ed59a8421aaa

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Clinical Biochemistry

1. Kevin J. Tracey
2. U. Andersson
3. R. Dhupar
4. Jon S. Cardinal
5. J. Klune
6. A. Tsung
7. Timothy R. Billiar

In Epidemiology

1. Kevin J. Tracey

In Molecular Biology

1. U. Andersson
2. Kevin J. Tracey
3. Bruce S. McEwen
4. Peter J. Gianaros
5. C. Deutschman

Citations in the corpus

• HMGB1 Is a Therapeutic Target for Sterile Inflammation and Infection (2011, background): “Endothelial cells Proangiogenic; upregulation of adhesion molecules (112, 113, 133, 134)”
• High mobility group box 1 protein interacts with multiple Toll-like receptors. (2006, background): “Although HMGB1 and LPS appear to initiate similar intracellular events, including activation of kinases such as p38, ERK1/2, and Akt and transcriptional factors including NF- B, that lead to production of proinflammatory cytokines, gene arrays demonstrated differences in expression profiles with eac”
• HMGB1: Endogenous Danger Signaling (2008, background): “These effects appear to be at least partially mediated by RAGE, which also appears to be upregulated in HMGB1stimulated endothelial cells (57,58).”