E. Cattaneo

Molecular Biology · 2000–2010

A researcher in the Science corpus: 9 papers; trusts 2 researchers and is trusted by 4 researchers here, as inferred from the citation graph.

Subfields

Whom E. Cattaneo trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. C. Zuccato
  2. M. Sheng

Who trusts E. Cattaneo overall

  1. S. Ghavami
  2. C. Zuccato
  3. A. Ardekani

Papers from E. Cattaneo's perspective

Ordered by how strongly the researchers E. Cattaneo trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Inhibition of mTOR induces autophagy and reduces toxicity of polyglutamine expansions in fly and mouse models of Huntington disease (Nature Genetics)
  2. Huntingtin interacts with REST/NRSF to modulate the transcription of NRSE-controlled neuronal genes (Nature Genetics)
  3. Suberoylanilide hydroxamic acid, a histone deacetylase inhibitor, ameliorates motor deficits in a mouse model of Huntington's disease (Proceedings of the National Academy of Sciences of the United States of America)
  4. Impairment of the ubiquitin-proteasome system by protein aggregation. (Science)
  5. Increased sensitivity to N-methyl-D-aspartate receptor-mediated excitotoxicity in a mouse model of Huntington's disease. (Neuron)
  6. The Huntington's disease protein interacts with p53 and CREB-binding protein and represses transcription. (Proceedings of the National Academy of Sciences of the United States of America)
  7. RNA interference improves motor and neuropathological abnormalities in a Huntington's disease mouse model. (Proceedings of the National Academy of Sciences of the United States of America)
  8. Reciprocal actions of REST and a microRNA promote neuronal identity (Proceedings of the National Academy of Sciences of the United States of America)
  9. Trehalose alleviates polyglutamine-mediated pathology in a mouse model of Huntington disease (Nature Medicine)
  10. Reversal of neuropathology and motor dysfunction in a conditional model of Huntington's disease. (Cell)

Their papers