C. Zuccato
Cellular and Molecular Neuroscience · 2001–2010
A researcher in the Science corpus: 5 papers; trusts 1 researcher and is trusted by 3 researchers here, as inferred from the citation graph.
Subfields
- Cellular and Molecular Neuroscience — 68 citations into it; #1,885 of 2,998 by trust here, #802 by citations
Whom C. Zuccato trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- Kenneth S. Kosik
- Sovan Sarkar
- E. Cattaneo
- A. Kazantsev
- G. Blander
- Ruth Geiss‐Friedlander
- J. Nithianantharajah
Who trusts C. Zuccato overall
- Siresha Bathina
- Akira Yoshii
- Mark P. Mattson
- E. Cattaneo
- Undurti N. Das
- Richard O'Brien
- D. Binder
- M. Valenza
- M. Constantine‐Paton
- S. Ghavami
Papers from C. Zuccato's perspective
Ordered by how strongly the researchers C. Zuccato trusts endorse each paper. Same corpus, a different order for every researcher.
- Inhibition of mTOR induces autophagy and reduces toxicity of polyglutamine expansions in fly and mouse models of Huntington disease (Nature Genetics)
- Suberoylanilide hydroxamic acid, a histone deacetylase inhibitor, ameliorates motor deficits in a mouse model of Huntington's disease (Proceedings of the National Academy of Sciences of the United States of America)
- Increased sensitivity to N-methyl-D-aspartate receptor-mediated excitotoxicity in a mouse model of Huntington's disease. (Neuron)
- Impairment of the ubiquitin-proteasome system by protein aggregation. (Science)
- The Huntington's disease protein interacts with p53 and CREB-binding protein and represses transcription. (Proceedings of the National Academy of Sciences of the United States of America)
- Huntingtin interacts with REST/NRSF to modulate the transcription of NRSE-controlled neuronal genes (Nature Genetics)
- Reversal of neuropathology and motor dysfunction in a conditional model of Huntington's disease. (Cell)
- Lithium induces autophagy by inhibiting inositol monophosphatase (Journal of Cell Biology)
- Trehalose alleviates polyglutamine-mediated pathology in a mouse model of Huntington disease (Nature Medicine)
- RNA interference improves motor and neuropathological abnormalities in a Huntington's disease mouse model. (Proceedings of the National Academy of Sciences of the United States of America)
Their papers
- Huntingtin interacts with REST/NRSF to modulate the transcription of NRSE-controlled neuronal genes (2003) — 979 citations
- Molecular mechanisms and potential therapeutical targets in Huntington's disease. (2010) — 850 citations
- Loss of Huntingtin-Mediated BDNF Gene Transcription in Huntington's Disease (2001) — 1,397 citations
- Normal huntingtin function: an alternative approach to Huntington's disease (2005) — 688 citations
- Brain-derived neurotrophic factor in neurodegenerative diseases (2009) — 925 citations