Philipp Oberdoerffer
Molecular Biology · 2008–2020
A researcher in the Science corpus: 4 papers; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 66 citations into it
Whom Philipp Oberdoerffer trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts Philipp Oberdoerffer overall
Papers from Philipp Oberdoerffer's perspective
Ordered by how strongly the researchers Philipp Oberdoerffer trusts endorse each paper. Same corpus, a different order for every researcher.
- Calorie restriction--the SIR2 connection. (Cell)
- Gene-expression profile of the ageing brain in mice (Nature Genetics)
- hSIR2(SIRT1) functions as an NAD-dependent p53 deacetylase. (Cell)
- Human SirT1 interacts with histone H1 and promotes formation of facultative heterochromatin. (Molecules and Cells)
- The SIRT1 Deacetylase Suppresses Intestinal Tumorigenesis and Colon Cancer Growth (PLoS ONE)
- Mammalian sirtuins--emerging roles in physiology, aging, and calorie restriction. (Genes & Development)
- Genomic instability and aging-like phenotype in the absence of mammalian SIRT6. (Cell)
- Sirt1 promotes fat mobilization in white adipocytes by repressing PPAR-γ (Nature)
- Augmented Wnt Signaling in a Mammalian Model of Accelerated Aging (Science)
- Modulation of NF‐κB‐dependent transcription and cell survival by the SIRT1 deacetylase (EMBO Journal)
Their papers
- DNA damage-induced alterations in chromatin contribute to genomic integrity and age-related changes in gene expression (2008) — 791 citations
- CTCF-promoted RNA polymerase II pausing links DNA methylation to splicing (2011) — 1,006 citations
- The Human Tumor Atlas Network: Charting Tumor Transitions across Space and Time at Single-Cell Resolution (2020) — 482 citations
- The SIRT1 Deacetylase Suppresses Intestinal Tumorigenesis and Colon Cancer Growth (2008) — 626 citations