Andrew N. Blackford

Molecular Biology · 2017

A researcher in the Science corpus: 1 paper; trusts 5 researchers and is trusted by 1 researcher here, as inferred from the citation graph.

Subfields

Whom Andrew N. Blackford trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Mark J. O'Connor
  2. C. Lukas
  3. Stephen P. Jackson
  4. C. Lord
  5. J. Bartek
  6. D. Hanahan
  7. Morgane Macheret
  8. J. Falck
  9. J. R. Chapman

Who trusts Andrew N. Blackford overall

  1. Ruixue Huang

Papers from Andrew N. Blackford's perspective

Ordered by how strongly the researchers Andrew N. Blackford trusts endorse each paper. Same corpus, a different order for every researcher.

  1. DNA damage activates ATM through intermolecular autophosphorylation and dimer dissociation (Nature)
  2. Conserved modes of recruitment of ATM, ATR and DNA-PKcs to sites of DNA damage (Nature)
  3. Hallmarks of cancer: the next generation. (Cell)
  4. MDC1 directly binds phosphorylated histone H2AX to regulate cellular responses to DNA double-strand breaks. (Cell)
  5. Activation of the DNA damage checkpoint and genomic instability in human precancerous lesions (Nature)
  6. Functional uncoupling of MCM helicase and DNA polymerase activities activates the ATR-dependent checkpoint. (Genes & Development)
  7. RNF168 ubiquitinates K13-15 on H2A/H2AX to drive DNA damage signaling. (Cell)
  8. 53BP1 nuclear bodies form around DNA lesions generated by mitotic transmission of chromosomes under replication stress (Nature Cell Biology)
  9. ATR prohibits replication catastrophe by preventing global exhaustion of RPA. (Cell)
  10. Requirement of the MRN complex for ATM activation by DNA damage (EMBO Journal)

Their papers