Jessica E. Bolden
Molecular Biology · 2006–2013
A researcher in the Science corpus: 3 papers; trusts 1 researcher and is trusted by 1 researcher here, as inferred from the citation graph.
Subfields
- Molecular Biology — 63 citations into it; #15,398 of 17,773 by trust here, #9,136 by citations
Whom Jessica E. Bolden trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts Jessica E. Bolden overall
- PA Marks
- E. Seto
- Ricky W. Johnstone
- Manel Esteller
- Peter A. Jones
- Xiang-Jiao Yang
- Manuel Rodríguez‐Paredes
- A. Kazantsev
- M. Haberland
Papers from Jessica E. Bolden's perspective
Ordered by how strongly the researchers Jessica E. Bolden trusts endorse each paper. Same corpus, a different order for every researcher.
- Deacetylation of p53 modulates its effect on cell growth and apoptosis (Nature)
- Clinical development of histone deacetylase inhibitors as anticancer agents. (Annual Review of Pharmacology and Toxicology)
- Aggresomes, inclusion bodies and protein aggregation. (Trends in Cell Biology)
- Acetylation of the C terminus of Ku70 by CBP and PCAF controls Bax-mediated apoptosis. (Molecules and Cells)
- Histone deacetylase inhibitor selectively induces p21WAF1 expression and gene-associated histone acetylation. (Proceedings of the National Academy of Sciences of the United States of America)
- Acetylation and deacetylation of non-histone proteins. (Gene)
- Life in the balance: how BH3-only proteins induce apoptosis. (Current Opinion in Cell Biology)
- Small-molecule inhibition of proteasome and aggresome function induces synergistic antitumor activity in multiple myeloma. (Proceedings of the National Academy of Sciences of the United States of America)
- HSP90 and the chaperoning of cancer (Nature Reviews. Cancer)
- Role of NF-κB in p53-mediated programmed cell death (Nature)
Their papers
- Anticancer activities of histone deacetylase inhibitors (2006) — 2,938 citations
- Non-cell-autonomous tumor suppression by p53 (2013) — 749 citations
- Control of the senescence-associated secretory phenotype by NF-κB promotes senescence and enhances chemosensitivity. (2011) — 978 citations