Marco J. Herold
Molecular Biology · 2014–2020
A researcher in the Science corpus: 4 papers; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 96 citations into it
Whom Marco J. Herold trusts
Their strongest outbound trust, strongest first, inferred from their citations.
- A. Strasser (in Molecular Biology)
Papers from Marco J. Herold's perspective
Ordered by how strongly the researchers Marco J. Herold trusts endorse each paper. Same corpus, a different order for every researcher.
- Ferroptosis as a p53-mediated activity during tumour suppression (Nature)
- Differential targeting of prosurvival Bcl-2 proteins by their BH3-only ligands allows complementary apoptotic function. (Molecules and Cells)
- p53 opens the mitochondrial permeability transition pore to trigger necrosis (Cell)
- Pyroptosis drives CD4 T-cell depletion in HIV-1 infection (Nature)
- An inhibitor of Bcl-2 family proteins induces regression of solid tumours (Nature)
- BH3 domains of BH3-only proteins differentially regulate Bax-mediated mitochondrial membrane permeabilization both directly and indirectly. (Molecules and Cells)
- In Vivo Activation of the p53 Pathway by Small-Molecule Antagonists of MDM2 (Science)
- Caspase functions in cell death and disease. (Cold Spring Harbor Perspectives in Biology)
- Obligate Role of Anti-Apoptotic MCL-1 in the Survival of Hematopoietic Stem Cells (Science)
- p53 induces differentiation of mouse embryonic stem cells by suppressing Nanog expression (Nature Cell Biology)
Their papers
- The MCL1 inhibitor S63845 is tolerable and effective in diverse cancer models (2016) — 971 citations
- Emerging connectivity of programmed cell death pathways and its physiological implications (2020) — 841 citations
- How does p53 induce apoptosis and how does this relate to p53-mediated tumour suppression? (2017) — 945 citations
- Apoptotic Caspases Suppress mtDNA-Induced STING-Mediated Type I IFN Production (2014) — 921 citations