Teresa Zelante
Infectious Diseases · 2007–2014
A researcher in the Science corpus: 4 papers; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Infectious Diseases — 12 citations into it
- Epidemiology — 8 citations into it
Whom Teresa Zelante trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Papers from Teresa Zelante's perspective
Ordered by how strongly the researchers Teresa Zelante trusts endorse each paper. Same corpus, a different order for every researcher.
- Surface phenotype and antigenic specificity of human interleukin 17–producing T helper memory cells (Nature Immunology)
- IL-23 drives a pathogenic T cell population that induces autoimmune inflammation
- TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells. (Immunity)
- A distinct lineage of CD4 T cells regulates tissue inflammation by producing interleukin 17 (Nature Immunology)
- Requirement of Interleukin 17 Receptor Signaling for Lung Cxc Chemokine and Granulocyte Colony-Stimulating Factor Expression, Neutrophil Recruitment, and Host Defense (Journal of Experimental Medicine)
- Induction of intestinal Th17 cells by segmented filamentous bacteria (Cell)
- Interleukin-23 Promotes a Distinct CD4 T Cell Activation State Characterized by the Production of Interleukin-17*
- Immunity to fungal infections (Nature reviews. Immunology)
- Ido expression by dendritic cells: tolerance and tryptophan catabolism (Nature reviews. Immunology)
- Interleukin-17 family members and inflammation. (Immunity)
Their papers
- Tryptophan catabolites from microbiota engage aryl hydrocarbon receptor and balance mucosal reactivity via interleukin-22. (2013) — 2,245 citations
- Aryl hydrocarbon receptor control of a disease tolerance defense pathway (2014) — 628 citations
- IRF4 Transcription Factor-Dependent CD11b+ Dendritic Cells in Human and Mouse Control Mucosal IL-17 Cytokine Responses (2013) — 816 citations
- IL‐23 and the Th17 pathway promote inflammation and impair antifungal immune resistance (2007) — 609 citations