A. Simon
Molecular Biology · Nature reviews. Drug discovery · 2009–2012
A researcher in the Science corpus: 3 papers; trusts 2 researchers and is trusted by 3 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 55 citations into it; #8,559 of 17,773 by trust here, #6,283 by citations
Whom A. Simon trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts A. Simon overall
Papers from A. Simon's perspective
Ordered by how strongly the researchers A. Simon trusts endorse each paper. Same corpus, a different order for every researcher.
- Gout-associated uric acid crystals activate the NALP3 inflammasome (Nature)
- Cryopyrin activates the inflammasome in response to toxins and ATP (Nature)
- The pyroptosome: a supramolecular assembly of ASC dimers mediating inflammatory cell death via caspase-1 activation (Cell Death and Differentiation)
- A frameshift mutation in NOD2 associated with susceptibility to Crohn's disease (Nature)
- Critical Role for Cryopyrin/Nalp3 in Activation of Caspase-1 in Response to Viral Infection and Double-stranded RNA*> (Journal of Biological Chemistry)
- NALP1 in vitiligo-associated multiple autoimmune disease. (New England Journal of Medicine)
- Chronic infantile neurological cutaneous and articular syndrome is caused by mutations in CIAS1, a gene highly expressed in polymorphonuclear cells and chondrocytes. (American Journal of Human Genetics)
- Reconstituted NALP1 inflammasome reveals two-step mechanism of caspase-1 activation. (Molecules and Cells)
- Association of NOD2 leucine-rich repeat variants with susceptibility to Crohn's disease (Nature)
- Bacterial RNA and small antiviral compounds activate caspase-1 through cryopyrin/Nalp3 (Nature)
Their papers
- Horror autoinflammaticus: the molecular pathophysiology of autoinflammatory disease (*). (2009) — 1,096 citations
- Mitochondrial reactive oxygen species promote production of proinflammatory cytokines and are elevated in TNFR1-associated periodic syndrome (TRAPS) (2011) — 844 citations
- Treating inflammation by blocking interleukin-1 in a broad spectrum of diseases (2012) — 1,758 citations