Prashanth T. Bhaskar
Molecular Biology · 2004–2013
A researcher in the Science corpus: 3 papers; trusts 1 researcher and is trusted by 1 researcher here, as inferred from the citation graph.
Subfields
- Molecular Biology — 77 citations into it; #15,807 of 17,773 by trust here, #8,182 by citations
Whom Prashanth T. Bhaskar trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts Prashanth T. Bhaskar overall
Papers from Prashanth T. Bhaskar's perspective
Ordered by how strongly the researchers Prashanth T. Bhaskar trusts endorse each paper. Same corpus, a different order for every researcher.
- Understanding the Warburg Effect: The Metabolic Requirements of Cell Proliferation (Science)
- Beyond aerobic glycolysis: Transformed cells can engage in glutamine metabolism that exceeds the requirement for protein and nucleotide synthesis (Proceedings of the National Academy of Sciences of the United States of America)
- Proapoptotic BAX and BAK: A Requisite Gateway to Mitochondrial Dysfunction and Death (Science)
- BH3-only proteins that bind pro-survival Bcl-2 family members fail to induce apoptosis in the absence of Bax and Bak. (Genes & Development)
- Inhibition of early apoptotic events by Akt/PKB is dependent on the first committed step of glycolysis and mitochondrial hexokinase. (Genes & Development)
- The mitochondrion in apoptosis: how Pandora's box opens (Nature reviews. Molecular cell biology)
- How cancer metabolism is tuned for proliferation and vulnerable to disruption (Nature)
- Analysis of lung tumor initiation and progression using conditional expression of oncogenic K-ras. (Genes & Development)
- The Key Role of Anaplerosis and Cataplerosis for Citric Acid Cycle Function* (Journal of Biological Chemistry)
- Functional genomics reveal that the serine synthesis pathway is essential in breast cancer
Their papers
- Hexokinase-mitochondria interaction mediated by Akt is required to inhibit apoptosis in the presence or absence of Bax and Bak. (2004) — 661 citations
- Hexokinase 2 is required for tumor initiation and maintenance and its systemic deletion is therapeutic in mouse models of cancer (2013) — 818 citations
- The two TORCs and Akt. (2007) — 847 citations