G. Milan

Molecular Biology · 2007–2015

A researcher in the Science corpus: 3 papers; trusts 2 researchers and is trusted by 0 researchers here, as inferred from the citation graph.

Subfields

Whom G. Milan trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. M. Sandri
  2. Jinghui Zhao
  3. J. Sacheck
  4. S. Lecker
  5. C. Mammucari
  6. N. Mizushima

Who trusts G. Milan overall

  1. I. Slaninová

Papers from G. Milan's perspective

Ordered by how strongly the researchers G. Milan trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Foxo transcription factors induce the atrophy-related ubiquitin ligase atrogin-1 and cause skeletal muscle atrophy. (Cell)
  2. The IGF-1/PI3K/Akt pathway prevents expression of muscle atrophy-induced ubiquitin ligases by inhibiting FOXO transcription factors. (Molecules and Cells)
  3. Multiple types of skeletal muscle atrophy involve a common program of changes in gene expression (The FASEB Journal)
  4. Trehalose, a Novel mTOR-independent Autophagy Enhancer, Accelerates the Clearance of Mutant Huntingtin and α-Synuclein* (Journal of Biological Chemistry)
  5. IKKbeta/NF-kappaB activation causes severe muscle wasting in mice. (Cell)
  6. Atrogin-1, a muscle-specific F-box protein highly expressed during muscle atrophy (Proceedings of the National Academy of Sciences of the United States of America)
  7. Rapid disuse and denervation atrophy involve transcriptional changes similar to those of muscle wasting during systemic diseases (The FASEB Journal)
  8. Protein degradation by the ubiquitin-proteasome pathway in normal and disease states. (Journal of the American Society of Nephrology)
  9. Autophagy in Health and Disease: A Double-Edged Sword (Science)
  10. Akt/mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivo (Nature Cell Biology)

Their papers