J. Sacheck

Molecular Biology · 2004–2007

A researcher in the Science corpus: 2 papers; trusts 4 researchers and is trusted by 2 researchers here, as inferred from the citation graph.

Subfields

Whom J. Sacheck trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. S. Lecker
  2. M. W. Pfaffl
  3. D. Glass
  4. M. Sandri
  5. A. Goldberg

Who trusts J. Sacheck overall

  1. M. Sandri
  2. C. Mammucari
  3. Jinghui Zhao
  4. G. Milan
  5. V. Romanello
  6. P. Bonaldo
  7. A. Goldberg
  8. S. Lecker
  9. S. Schiaffino
  10. J. Brault

Papers from J. Sacheck's perspective

Ordered by how strongly the researchers J. Sacheck trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Atrogin-1, a muscle-specific F-box protein highly expressed during muscle atrophy (Proceedings of the National Academy of Sciences of the United States of America)
  2. Mediation of IGF-1-induced skeletal myotube hypertrophy by PI(3)K/Akt/mTOR and PI(3)K/Akt/GSK3 pathways (Nature Cell Biology)
  3. Akt/mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivo (Nature Cell Biology)
  4. Functional discovery via a compendium of expression profiles. (Cell)
  5. An integrated stress response regulates amino acid metabolism and resistance to oxidative stress. (Molecules and Cells)
  6. Foxo transcription factors induce the atrophy-related ubiquitin ligase atrogin-1 and cause skeletal muscle atrophy. (Cell)
  7. The IGF-1/PI3K/Akt pathway prevents expression of muscle atrophy-induced ubiquitin ligases by inhibiting FOXO transcription factors. (Molecules and Cells)
  8. Identification of Ubiquitin Ligases Required for Skeletal Muscle Atrophy (Science)
  9. Signalling pathways that mediate skeletal muscle hypertrophy and atrophy (Nature Cell Biology)
  10. Transcriptional co-activator PGC-1 alpha drives the formation of slow-twitch muscle fibres. (Nature)

Their papers