Matthias Peter
Science · 2002–2016
A researcher in the Science corpus: 7 papers; trusts 0 researchers and is trusted by 8 researchers here, as inferred from the citation graph.
Who trusts Matthias Peter overall
Papers from Matthias Peter's perspective
Ordered by how strongly the researchers Matthias Peter trusts endorse each paper. Same corpus, a different order for every researcher.
- PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1 (Nature Cell Biology)
- p62/SQSTM1 Binds Directly to Atg8/LC3 to Facilitate Degradation of Ubiquitinated Protein Aggregates by Autophagy* (Journal of Biological Chemistry)
- Autophagosome formation: core machinery and adaptations (Nature Cell Biology)
- Mature ribosomes are selectively degraded upon starvation by an autophagy pathway requiring the Ubp3p/Bre5p ubiquitin protease (Nature Cell Biology)
- Nix is a selective autophagy receptor for mitochondrial clearance (EMBO Reports)
- Tor-Mediated Induction of Autophagy via an Apg1 Protein Kinase Complex (Journal of Cell Biology)
- Aggresomes, inclusion bodies and protein aggregation. (Trends in Cell Biology)
- HDAC6 and Microtubules Are Required for Autophagic Degradation of Aggregated Huntingtin* (Journal of Biological Chemistry)
- Impairment of the ubiquitin-proteasome system by protein aggregation. (Science)
- Autophagy Inhibition Compromises Degradation of Ubiquitin-Proteasome Pathway Substrates (Molecules and Cells)
Their papers
- Mature ribosomes are selectively degraded upon starvation by an autophagy pathway requiring the Ubp3p/Bre5p ubiquitin protease (2008) — 742 citations
- Global Mapping of the Yeast Genetic Interaction Network (2004) — 2,147 citations
- Ubiquitin-Binding Domains in Y-Family Polymerases Regulate Translesion Synthesis (2005) — 733 citations
- CellCognition: time-resolved phenotype annotation in high-throughput live cell imaging (2010) — 381 citations
- Selective autophagy: ubiquitin-mediated recognition and beyond (2010) — 649 citations
- Translocon component Sec62 acts in endoplasmic reticulum turnover during stress recovery (2016)
- Cell cycle‐dependent nuclear export of Cdh1p may contribute to the inactivation of APC/CCdh1 (2002) — 98 citations