Peng Huang

Molecular Biology · Nature reviews. Drug discovery · 2000–2018

A researcher in the Science corpus: 10 papers; trusts 5 researchers and is trusted by 8 researchers here, as inferred from the citation graph.

Subfields

• Molecular Biology — 137 citations into it; #5,613 of 17,773 by trust here, #2,317 by citations
• Oncology — 40 citations into it
• Biochemistry — 9 citations into it
• Endocrine and Autonomic Systems — 8 citations into it
• Plant Science — 8 citations into it

Whom Peng Huang trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. X. Wang

Who trusts Peng Huang overall

1. D. Trachootham
2. Yongqiang Chen
3. H. Pelicano
4. M. Azad
5. David B. Graves

Papers from Peng Huang's perspective

Ordered by how strongly the researchers Peng Huang trusts endorse each paper. Same corpus, a different order for every researcher.

1. BCL-2, BCL-X(L) sequester BH3 domain-only molecules preventing BAX- and BAK-mediated mitochondrial apoptosis. (Molecules and Cells)
2. Why do cancers have high aerobic glycolysis? (Nature Reviews. Cancer)
3. ROS stress in cancer cells and therapeutic implications. (Drug resistance updates)
4. Attenuation of LDH-A expression uncovers a link between glycolysis, mitochondrial physiology, and tumor maintenance. (Cancer Cell)
5. Proliferation, cell cycle and apoptosis in cancer (Nature)
6. TIGAR, a p53-inducible regulator of glycolysis and apoptosis. (Cell)
7. Cancer metabolism: facts, fantasy, and fiction. (Biochemical and Biophysical Research Communications - BBRC)
8. Cellular response to oxidative stress: Signaling for suicide and survival * (Journal of Cellular Physiology)
9. BAD and glucokinase reside in a mitochondrial complex that integrates glycolysis and apoptosis (Nature)
10. Intrinsic resistance of tumorigenic breast cancer cells to chemotherapy. (Journal of the National Cancer Institute)

Their papers

• Selective killing of oncogenically transformed cells through a ROS-mediated mechanism by beta-phenylethyl isothiocyanate. (2006) — 1,121 citations
• Glycolysis inhibition for anticancer treatment (2006) — 1,515 citations
• BAP1 links metabolic regulation of ferroptosis to tumor suppression (2018) — 826 citations
• Inhibition of glycolysis in cancer cells: a novel strategy to overcome drug resistance associated with mitochondrial respiratory defect and hypoxia. (2005) — 860 citations
• Superoxide dismutase as a target for the selective killing of cancer cells (2000) — 869 citations
• Targeting cancer cells by ROS-mediated mechanisms: a radical therapeutic approach? (2009) — 5,133 citations
• Redox Regulation of Cell Survival (2008) — 1,755 citations
• ROS stress in cancer cells and therapeutic implications. (2004) — 1,967 citations
• Mitochondrial defects in cancer (2002) — 613 citations
• The circadian gene Period2 plays an important role in tumor suppression and DNA damage response in vivo. (2002) — 1,316 citations