H. Pelicano
Molecular Biology · 2002–2006
A researcher in the Science corpus: 5 papers; trusts 2 researchers and is trusted by 5 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 32 citations into it; #9,027 of 17,773 by trust here, #5,504 by citations
- Cancer Research — 16 citations into it; #794 of 1,780 by trust here, #917 by citations
- Endocrine and Autonomic Systems — 8 citations into it
- Plant Science — 7 citations into it
Whom H. Pelicano trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts H. Pelicano overall
- M. Azad
- Yongqiang Chen
- David B. Graves
- D. Trachootham
- G. Barrera
- G. J. Yoshida
- M. López-Lázaro
- V. Gogvadze
- J. Chiche
- M. Brahimi-Horn
Papers from H. Pelicano's perspective
Ordered by how strongly the researchers H. Pelicano trusts endorse each paper. Same corpus, a different order for every researcher.
- BCL-2, BCL-X(L) sequester BH3 domain-only molecules preventing BAX- and BAK-mediated mitochondrial apoptosis. (Molecules and Cells)
- Cancer metabolism: facts, fantasy, and fiction. (Biochemical and Biophysical Research Communications - BBRC)
- Proliferation, cell cycle and apoptosis in cancer (Nature)
- BAD and glucokinase reside in a mitochondrial complex that integrates glycolysis and apoptosis (Nature)
- Akt-Directed Glucose Metabolism Can Prevent Bax Conformation Change and Promote Growth Factor-Independent Survival (Molecular and Cellular Biology)
- Akt Stimulates Aerobic Glycolysis in Cancer Cells (Cancer Research)
- Inhibition of early apoptotic events by Akt/PKB is dependent on the first committed step of glycolysis and mitochondrial hexokinase. (Genes & Development)
- ROS stress in cancer cells and therapeutic implications. (Drug resistance updates)
- Alkylating DNA damage stimulates a regulated form of necrotic cell death. (Genes & Development)
- Cellular response to oxidative stress: Signaling for suicide and survival * (Journal of Cellular Physiology)
Their papers
- Selective killing of oncogenically transformed cells through a ROS-mediated mechanism by beta-phenylethyl isothiocyanate. (2006) — 1,121 citations
- Glycolysis inhibition for anticancer treatment (2006) — 1,515 citations
- Inhibition of glycolysis in cancer cells: a novel strategy to overcome drug resistance associated with mitochondrial respiratory defect and hypoxia. (2005) — 860 citations
- ROS stress in cancer cells and therapeutic implications. (2004) — 1,967 citations
- The circadian gene Period2 plays an important role in tumor suppression and DNA damage response in vivo. (2002) — 1,316 citations