M. Giuliani

Molecular Biology · 2014

A researcher in the Science corpus: 1 paper; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.

Subfields

Whom M. Giuliani trusts

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Masayuki Yamamoto (in Molecular Biology)

Papers from M. Giuliani's perspective

Ordered by how strongly the researchers M. Giuliani trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Oxidative Stress Sensor Keap1 Functions as an Adaptor for Cul3-Based E3 Ligase To Regulate Proteasomal Degradation of Nrf2 (Molecular and Cellular Biology)
  2. Structure of the Cul1–Rbx1–Skp1–F boxSkp2 SCF ubiquitin ligase complex (Nature)
  3. Direct evidence that sulfhydryl groups of Keap1 are the sensors regulating induction of phase 2 enzymes that protect against carcinogens and oxidants (Proceedings of the National Academy of Sciences of the United States of America)
  4. BTB Protein Keap1 Targets Antioxidant Transcription Factor Nrf2 for Ubiquitination by the Cullin 3-Roc1 Ligase (Molecular and Cellular Biology)
  5. Enhanced Expression of the Transcription Factor Nrf2 by Cancer Chemopreventive Agents: Role of Antioxidant Response Element-Like Sequences in the nrf2 Promoter (Molecular and Cellular Biology)
  6. Cell survival responses to environmental stresses via the Keap1-Nrf2-ARE pathway. (Annual Review of Pharmacology and Toxicology)
  7. Keap1 Is a Redox-Regulated Substrate Adaptor Protein for a Cul3-Dependent Ubiquitin Ligase Complex (Molecular and Cellular Biology)
  8. HIF-1, O(2), and the 3 PHDs: how animal cells signal hypoxia to the nucleus. (Cell)
  9. High sensitivity of Nrf2 knockout mice to acetaminophen hepatotoxicity associated with decreased expression of ARE-regulated drug metabolizing enzymes and antioxidant genes. (Toxicological Sciences)
  10. Nrf2-Keap1 defines a physiologically important stress response mechanism. (Trends in Molecular Medicine)

Their papers