F. Kamenetz
Physiology · 2003
A researcher in the Science corpus: 1 paper; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Physiology — 11 citations into it
- Computational Theory and Mathematics — 6 citations into it
Whom F. Kamenetz trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts F. Kamenetz overall
Papers from F. Kamenetz's perspective
Ordered by how strongly the researchers F. Kamenetz trusts endorse each paper. Same corpus, a different order for every researcher.
- Alzheimer's Disease Is a Synaptic Failure (Science)
- Driving AMPA receptors into synapses by LTP and CaMKII: requirement for GluR1 and PDZ domain interaction. (Science)
- Kinesin-mediated axonal transport of a membrane compartment containing β-secretase and presenilin-1 requires APP (Nature)
- Tau Suppression in a Neurodegenerative Mouse Model Improves Memory Function (Science)
- Neuropathological stageing of Alzheimer-related changes
- Opposing Activities Protect Against Age-Onset Proteotoxicity (Science)
- Activity level controls postsynaptic composition and signaling via the ubiquitin-proteasome system (Nature Neuroscience)
- Short-term synaptic plasticity.
- Amyloid fibrillogenesis: themes and variations. (Current Opinion in Structural Biology)
- Common Structure of Soluble Amyloid Oligomers Implies Common Mechanism of Pathogenesis (Science)
Their papers
- APP processing and synaptic function. (2003) — 1,647 citations