The SLC2 family of facilitated hexose and polyol transporters
By Marc Uldry, B. Thorens. Pflügers Archiv, 2004
513 citations recorded by the source corpus.
Subfields: Molecular Biology, Rheumatology, Surgery
View Paper: https://www.semanticscholar.org/paper/f85c51f051a3e91a40dbf05a480294eac0655cd5
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Citations in the corpus
- Na+-d-glucose Cotransporter SGLT1 is Pivotal for Intestinal Glucose Absorption and Glucose-Dependent Incretin Secretion (2011, background): “The primary transporters that mediate transcellular movements of D-glucose in small intestine have been identified and include the Na-D-glucose cotransporter SGLT1 and GLUT2 (3,4).”
- Supply and Demand in Cerebral Energy Metabolism: The Role of Nutrient Transporters (2007, background): “Interestingly, peripheral sites of GLUT3 expression include sperm, platelets, placenta, and certain human gliomas; all cell types that exhibit high rates of glucose metabolism (see Joost and Thorens, 2001; Uldry and Thorens, 2004; Wood and Trayhurn, 2003).”
- Glucose transporters in the 21st Century. (2010, background): “The cloning of cDNA encoding the red cell glucose transporter (Glut1) occurred in 1985 (29), and 13 related members of the SLC2A (Glut) protein family have subsequently been identified in the human (41).”
- SLC2A9 is a newly identified urate transporter influencing serum urate concentration, urate excretion and gout (2008, background): “SLC2A9 shows greatest sequence similarity to the fructose transporter SLC2A5, a class II transporte”
- The SLC2 (GLUT) Family of Membrane Transporters (2013, unknown): “The transport of monosaccharides, polyols, and other small carbon compounds across the membranes of eukaryotic cells is mediated by members of the GLUT family of integral membrane proteins that are encoded by the SLC2 genes and are members of the major facilitator superfamily (MFS) (see Table 1) [re”
- Recent advances in 2D and 3D in vitro systems using primary hepatocytes, alternative hepatocyte sources and non-parenchymal liver cells and their use in investigating mechanisms of hepatotoxicity, cell signaling and ADME (2013, unknown): “8 lM) (Uldry and Thorens 2004).”