TLRs, NLRs and RLRs: a trinity of pathogen sensors that co-operate in innate immunity.
By E. Creagh, Luke A. O'Neill. Trends in immunology, 2006
805 citations recorded by the source corpus.
Subfields: Epidemiology, Immunology
View Paper: https://www.semanticscholar.org/paper/f1415bd29eb4984410e2be046f871f76c5ae456b
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Citations in the corpus
- Cytokines in Inflammatory Disease (2019, background): “Upon their activation, PRR transduces signals intracellularly [12], e.”
- Neutrophil Elastase, Proteinase 3, and Cathepsin G as Therapeutic Targets in Human Diseases (2010, background): “leased by injured tissues (Akira et al., 2006; Creagh and O’Neill, 2006; Brikos and O’Neill, 2008; Buchanan et al., 2009).”
- Resolution of inflammation: state of the art, definitions and terms (2006, background): “The initiation of the inflammatory response during infection or in response to sterile tissue injury involves two families of receptors, the toll-like receptors (TLRs) and nod-like receptors (NLRs) (11).”
- Tumour necrosis factor and cancer (2009, background): “We now realize that Coley's mixed toxins must have been powerful stimulants of TLR”