DC-SIGN: escape mechanism for pathogens

By Y. Kooyk, Teunis B. Geijtenbeek. Nature reviews. Immunology, 2003

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Virus Entry: Open Sesame (2006, neutral): “Similar dendritic-cell-mediated trans-infection routes have been proposed for a number of other viruses, including herpes, filo-, and flaviviruses (van Kooyk and Geijtenbeek, 2003) .”
Genes and Molecules of Lactobacilli Supporting Probiotic Action (2008, background): “Certain pathogens also use DC-SIGN as an escape mechanism for immune surveillance and the induction of immunosuppressive effects (257).”
Host interactions of probiotic bacterial surface molecules: comparison with commensals and pathogens (2010, background): “This is especially interesting, as DC-SIGN is known to interact with various structurally diverse glycosylated ligands of pathogens and thereby has a key role in the final host response against the encountered microorganis”
The Continuing Challenges of Leprosy (2006, background): “ment of DC-SIGN may also inhibit TLR signaling (422).”
The surface protein TIGIT suppresses T cell activation by promoting the generation of mature immunoregulatory dendritic cells (2009, background): “Other proteins, including C-type lectins such as DC-SIGN and mannose receptor, modify DCs by inhibiting TLR-driven maturation and enhancing IL-10 productio”