Intestinal epithelial responses to enteric pathogens: effects on the tight junction barrier, ion transport, and inflammation

By J. Berkes, V. Viswanathan, S. Savkovic, G. Hecht. Gut, 2003

613 citations recorded by the source corpus.

Subfields: Molecular Biology, Molecular Medicine, Neurology

View Paper: https://www.semanticscholar.org/paper/e659300c353d75d7ca54b0273ac902812dc3e692

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In Molecular Biology

1. J. Cryan
2. Ted G. Dinan
3. G. Clarke
4. P. Kennedy
5. J. R. Kelly
6. R. Lynch
7. P. Cossart
8. S. Bischoff

In Molecular Medicine

1. P. Cossart
2. D. Ribet

In Neurology

1. R. Lynch
2. P. Cossart
3. E. Schneeberger
4. Katherine R. Groschwitz
5. S. Hogan
6. D. Ribet

Citations in the corpus

• How bacterial pathogens colonize their hosts and invade deeper tissues. (2015, methodology): “458 Finally, triggering of inflammation, in addition to its role on the microbiota discussed 459 above, was also proposed as a tool used by pathogens for host barriers disruption [89].”
• Breaking down the barriers: the gut microbiome, intestinal permeability and stress-related psychiatric disorders (2015, background): “Several enteric pathogens have been shown to have a detrimental effect on the intestinal barrier (Paesold et al., 2002; Berkes et al., 2003; Flynn and Buret, 2008) which can occur via paracellular (Ferrier et al., 2003; Wu et al., 2011) or transcellular routes (Kalischuk et al., 2009).”
• The tight junction: a multifunctional complex. (2004, background): “For the effect of enteric pathogens on TJ barrier function, see a recent review on this topic (22).”
• Intestinal permeability – a new target for disease prevention and therapy (2014, background): “Enteric pathogens often gain access to the body by altering the structure and function of tight junctions to increase permeability of the barrier via the secretion of proteases, which can cleave tight junction proteins or by altering the cytoskeleton [116].”
• Intestinal barrier function: molecular regulation and disease pathogenesis. (2009, background): “The dynamic and complex interactions between enteric pathogens and the intestinal epithelium often leads to disturbances in the intestinal barrier, altered fluid and electrolyte transport, and the induction of an inflammatory response.(162) Enteric pathogens can disrupt the intestinal barrier direct”
• New Insights on Classification, Identification, and Clinical Relevance of Blastocystis spp (2008, background): “inflammation (27), and since Blastocystis is a noninvasive par-”