Dendritic-cell control of pathogen-driven T-cell polarization

By M. Kapsenberg. Nature reviews. Immunology, 2003

1,576 citations recorded by the source corpus.

View Paper: https://www.semanticscholar.org/paper/d0f872721da5bd2a752a9f4a08ca7ff95382dbcc

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In Immunology

Citations in the corpus

Inflammatory mediators are insufficient for full dendritic cell activation and promote expansion of CD4+ T cell populations lacking helper function (2005, background): “However, it is becoming increasingly clear that activation is not a linear process and that the nature, intensity and/or duration of the activation signal are important in determining the effector function of DC”
An immunomodulatory molecule of symbiotic bacteria directs maturation of the host immune system. (2005, background): “SA Is Specifically Recognized by Dendritic Cells, ith Consequent Cell Activation ll CD4+ T cell reactions require instruction to T cells y APCs (Kidd, 2003; Kapsenberg, 2003).”
IL-10: The Master Regulator of Immunity to Infection (2008, background): “However, under certain conditions (typically following exposure to IL-10 or TGF- ) DC can initiate the development of Treg that limit these effector responses (39, 40).”
Dendritic cells in a mature age (2006, background): “DC maturation and the class of immunity Much has been written about the ability of DCs to direct different classes of immunit”
TLR Signaling Tailors Innate Immune Responses in Human Microglia and Astrocytes1 (2005, background): “Nonetheless, the polarization of immune responses has been shown to be a complex process, and factors such as Ag dose and coreceptors may also play an important role (40, 41).”
Regulation of immune responses by L-arginine metabolism (2005, background): “For example, dendritic cells (DCs), such as Langerhans cells in the skin, are strategically placed at the site of antigen entry, and they express a range of molecules that aid in priming naive T cell”