Ephrin-B2 controls VEGF-induced angiogenesis and lymphangiogenesis
By Yingdi Wang, Masanori Nakayama, M. Pitulescu, Tim S. Schmidt, M. Bochenek, Akira Sakakibara, Susanne Adams, A. Davy, U. Deutsch, U. Lüthi and 5 more. Nature, 2010
1,251 citations recorded by the source corpus.
Subfields: Cell Biology, Cellular and Molecular Neuroscience, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/cdea8d519ef0656a60b809ea0f2e7fe42816fae3
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Citations in the corpus
- Tumor angiogenesis: molecular pathways and therapeutic targets (2011, background): “Receptor clustering augments ligand binding, which triggers downstream signaling intermediates and often leads to receptor internalization or recycling to determine the magnitude and kinetics of the signaling respons”
- VE-cadherin and endothelial adherens junctions: active guardians of vascular integrity. (2013, background): “Whether ephrin B2 may interact with VE-cadherin in this process is unknown (Nakayama et al., 2013; Wang et al., 2010).”
- Signal transduction by vascular endothelial growth factor receptors. (2012, background): “VEGFR3 internalization and signaling involving RAC1, AKT, and ERK is regulated by ephrin B2 (Wang et al. 2010).”
- Basic and therapeutic aspects of angiogenesis. (2011, neutral): “ECs lacking ephrin-B2 reverse signaling are unable to internalize VEGFR2 and VEGFR3 and cannot transmit VEGF signals properly, together impairing sprouting (Sawamiphak et al., 2010; Wang et al., 2010).”
- Molecular control of endothelial cell behaviour during blood vessel morphogenesis (2011, background): “For example, recent data suggests that ephrin B2 may directly interact with VEGFRs to regulate VEGF signallin”