Diabetes and endothelial dysfunction: a clinical perspective.

By J. Callés-Escandon, M. Cipolla. Endocrine reviews, 2001

690 citations recorded by the source corpus.

Subfields: Cardiology and Cardiovascular Medicine, Physiology

View Paper: https://www.semanticscholar.org/paper/bd135d17c361024a11d00d4ae1de9362477b6727

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In Physiology

1. L. Liaudet
2. P. Pacher
3. B. Ahrén
4. M. Quon
5. David B. Graves
6. Rafael Radi

Citations in the corpus

• Effects of glucagon-like peptide-1 on endothelial function in type 2 diabetes patients with stable coronary artery disease. (2004, background): “It is well known that type 2 diabetes subjects are characterized by impaired endothelial-dependent relaxation, which may be a consequence of a disturbed synthesis or increased destruction of NO or a combination thereof (8).”
• Diabetic cardiomyopathy: evidence, mechanisms, and therapeutic implications. (2004, background): “(Endocrine Reviews 25: 543–567, 2004) I. Introduction II.”
• The Vascular Endothelium and Human Diseases (2013, background): “It was shown that, in normal subjects, insulin is able to induce a dose-dependent increase in lower limb blood flow by reducing vascular resistance in skeletal muscle (82), mainly by vasodilating the microcirculation (83).”
• Nitric oxide and peroxynitrite in health and disease. (2007, background): “Diabetes, and even prediabetes, is characterized by endothelial dysfunction, which precedes and predicts, as well as predisposes, for consequent, more severe vascular alteration (165, 169, 1242).”
• Cardiovascular actions of insulin. (2007, background): “Genetic contributions to defective insulin signaling and NO activity in the vasculature are likely to contribute to diminished vascular actions of insulin that play a role in the pathogenesis of insulin resistance and endothelial dysfunction per se in type 2 diabetes (263) and obesity (382).”
• Endothelial Dysfunction: Cardiovascular Risk Factors, Therapy, and Outcome (2005, neutral): “Irrespective of whether endothelial cell dysfunction is a cause or a consequence of vascular injury in type 1 diabetes, it is hoped that therapeutic efforts aimed at restoring endothelial cell function to normal will affect the natural history of vasculopathy in type 1 diabetes (Calles-Escandon and ”
• Metabolism and function of coenzyme Q. (2004, background): “One of the characteristic events in many diseases, such as diabetes and cardiovascular diseases, is endothelial dysfunction of the arteries which has serious consequences [91].”