Therapeutic manipulation of the enteric microflora in inflammatory bowel diseases: antibiotics, probiotics, and prebiotics.
By Balfour Sartor. Gastroenterology, 2004
1,138 citations recorded by the source corpus.
Subfields: Food Science, Genetics, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/b13739f29e8fb1e7c108d3fe827572a18cad29e5
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- David S. Wishart
- Markus R. Wenk
Citations in the corpus
- Reduced diversity of faecal microbiota in Crohn’s disease revealed by a metagenomic approach (2005, background): “Although the cause of inflammatory bowel disease remains unknown, the indigenous intestinal microbiota is considered a major if not the main trigger of inflammation, both in animal models and in humans.”
- Inulin: Properties, health benefits and food applications. (2016, background): “IBD is usually considered as western world disease, and its incidence has increased significantly during the past few decades (Sartor, 2004).”
- Connecting dysbiosis, bile-acid dysmetabolism and gut inflammation in inflammatory bowel diseases (2012, methodology): “For each sample, experiments were halted at different time points (0 min, 30 min, 180 min and 12 h) using three steps: (1) addition of 100 ml of 0.”
- Impact of the Gut Microbiota on Intestinal Immunity Mediated by Tryptophan Metabolism (2018, background): “Along with reduced CYP11A1 expression (Coste et al., 2007) and decreased glucocorticoid production (Huang et al., 2014), disorders of intestinal steroidogenesis have been associated with IBD.”
- Effects of dietary chitosan oligosaccharides and Bacillus coagulans on the growth, innate immunity and resistance of koi (Cyprinus carpio koi) (2012, background): “The role of probiotics in modulating the immune system has been extensively investigated and reviewed in humans and animals (Fooks et al., 1999; Galdeano and Perdigon, 2006; Nayak, 2010; Sartor, 2004).”
- Gut microbiome-host interactions in health and disease (2011, background): “This implies that each host has a unique biological relationship with its gut microbiota [22,23], and by definition that this influences an individual’s risk of disease.”