Induction of plasticity in the human motor cortex by paired associative stimulation.

By K. Stefan, E. Kunesch, Leonardo G. Cohen, R. Benecke, J. Classen. Brain : a journal of neurology, 2000

1,468 citations recorded by the source corpus.

Subfields: Cognitive Neuroscience, Neurology

View Paper: https://www.semanticscholar.org/paper/a6a124ce515058cc844097b04c7950b9f490bea8

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Citations in the corpus

Differential Modulation of Motor Cortical Plasticity and Excitability in Early and Late Phases of Human Motor Learning (2007, methodology): “7A), the PAS protocols produced the expected effects of MEP facilitation after PAS25 and suppression after PAS10 (Stefan et al., 2000; Wolters et al., 2003).”
Basic principles of transcranial magnetic stimulation (TMS) and repetitive TMS (rTMS). (2015, background): “© 201 interact to reshape local representational cortical patterns [41].”
Enhancement of human cognitive performance using transcranial magnetic stimulation (TMS) (2013, background): “For example, repeated co-stimulation of the median nerve in the forearm and motor cortex can potentiate subsequent MEPs evoked by TMS (Stefan et al., 2000; Wolters et al., 2003).”
Partially non‐linear stimulation intensity‐dependent effects of direct current stimulation on motor cortex excitability in humans (2013, neutral): “…for this delayed effect so far, although it has been observed in animal studies, and also for other non-invasive plasticity induction protocols in humans (Bindman et al. 1964; Bi & Poo, 1998; Stefan et al. 2000) and under lorazepam-reinforcing GABAergic contribution for tDCS (Nitsche et al. 2004b).”
Plasticity in the human central nervous system. (2006, methodology): “For example, peripheral stimulation of the right median nerve can be followed by TMS directed at the hand representation area in contralateral primary motor cortex (M1), at a latency determined by the time-lag in evoking an M1 cortical potential via activation of somatosensory cortex (Stefan et al.,”
Pharmacological approach to the mechanisms of transcranial DC-stimulation-induced after-effects of human motor cortex excitability. (2002, methodology): “Asterisks indicate signi®cant changes of the evoked potentials after tDCS as compared with the baseline values that are displayed as 100% (Student's t-test, P < 0.05). term sensory stimulation or paired somatosensory and TMS (Hamdy et al., 1998; Ziemann et al., 1998b; Stefan et al., 2000).”
Inter-individual variability in response to non-invasive brain stimulation paradigms. (2014, positive): “The most established protocols to increase cortical excitability (by recent citation records) are excitatory paired associative stimulation (PAS) [10], anodal transcranial direct current stimulation (AtDCS) [3] and intermittent theta burst stimulation (iTBS) [11].”
A temporally asymmetric Hebbian rule governing plasticity in the human motor cortex. (2003, methodology): “The physiological properties of the PAS(25ms)-induced increase of cortical excitability fulfill necessary criteria of LTP (Stefan et al. 2000; Stefan et al. 2002).”
Motor learning elicited by voluntary drive. (2003, methodology): “Recent studies demonstrated that somatosensory input in the form of peripheral nerve stimulation results in functional changes in corticomotor excitability (Ridding et al., 2000; Stefan et al., 2000; Kaelin-Lang et al., 2003).”
A common polymorphism in the brain‐derived neurotrophic factor gene (BDNF) modulates human cortical plasticity and the response to rTMS (2008, methodology): “Effects in the ADM are also variable: Stefan et al. (2000) originally reported that there was no significant difference in the facilitation of APB and ADM, but others have suggested that effects in ADM are generally smaller than in APB, consistent with a topographic specificity of PAS.”
Non-invasive electrical and magnetic stimulation of the brain, spinal cord, roots and peripheral nerves: Basic principles and procedures for routine clinical and research application. An updated report from an I.F.C.N. Committee (2015, methodology): “Usually, PAS is delivered at a relatively low frequency (0.01–0.25 Hz) (Stefan et al., 2000; Ziemann, 2004; Müller et al., 2007), but some authors showed long-lasting changes in motor cortex excitability following PAS25 applied at 5 Hz (Quartarone et al., 2006).”
Functional potential in chronic stroke patients depends on corticospinal tract integrity. (2006, background): “Priming the ipsilesional cortex, as an adjuvant therapy, may improve functional outcomes in this group (Stefan et al., 2000; Stinear and Byblow, 2004b; Fregni et al., 2005; Huang et al., 2005; Hummel et al., 2005; Khedr et al., 2005; Mansur et al., 2005; Takeuchi et al., 2005; Smith and Staines, 200”