Exogenous Induction of Cerebral ß-Amyloidogenesis Is Governed by Agent and Host

By Melanie Meyer‐Luehmann, Janaky Coomaraswamy, T. Bolmont, S. Kaeser, C. Schaefer, E. Kilger, Anton Neuenschwander, D. Abramowski, P. Frey, A. Jaton and 8 more. Science, 2006

983 citations recorded by the source corpus.

Subfields: Molecular Biology, Neurology, Physiology

View Paper: https://www.semanticscholar.org/paper/a2b5a9210ee24e3b1dac162ef13f1ffa9a05a4ad

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In Molecular Biology

1. A. Paravastu
2. Richard Leapman
3. Oliver D. King
4. M. Murphy
5. H. Levine
6. R. Tycko
7. John Q. Trojanowski
8. Wai-Ming Yau

In Neurology

1. V. Lee
2. Jing L Guo
3. John Q. Trojanowski
4. E. Masliah
5. D. Holtzman
6. Bin Zhang
7. Oliver D. King
8. J. Shorter

In Physiology

1. D. Holtzman
2. V. Lee
3. Jing L Guo
4. John Q. Trojanowski
5. A. Paravastu
6. Richard Leapman
7. Bin Zhang
8. E. Masliah

Citations in the corpus

• Inclusion formation and neuronal cell death through neuron-to-neuron transmission of α-synuclein (2009, background): “A similar mechanism of pathological propagation has been suggested in a mouse model of Alzheimer’s disease, in which injection of brain extracts from human patients with Alzheimer’s disease or from older amyloid precursor protein (APP) transgenic mice with plaque pathology induced amyloid deposition”
• The tip of the iceberg: RNA-binding proteins with prion-like domains in neurodegenerative disease (2012, background): “(2012), doi:10.1016/j.brainres.2012.01.016 genomes (~100–400 per genome), but rare in prokaryotes (Michelitsch and Weissman, 2000).”
• Rapid appearance and local toxicity of amyloid-β plaques in a mouse model of Alzheimer’s disease (2008, background): “It is possible that submicroscopic nuclei may be present in the cortex 23 , perhaps related to the recently reported amyloid-β oligomeric forms 24,25 , as precursors to this sudden growth.”