Genomic binding sites of the yeast cell-cycle transcription factors SBF and MBF

By V. Iyer, C. Horak, C. Scafe, David Botstein, Michael Snyder, Patrick O. Brown. Nature, 2001

1,151 citations recorded by the source corpus.

View Paper: https://www.semanticscholar.org/paper/97be6194399041a2474ab41bcbac75c6ae9ee639

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Molecular Biology

Citations in the corpus

BAC TransgeneOmics (2008, methodology): “Methods for genome-wide mapping of DNA binding sites based on chromatin immunopurification (ChIP) coupled with microarray analysis (ChIP-chip”
Mapping of genetic and epigenetic regulatory networks using microarrays (2005, background): “Such complete maps have been generated in Saccharomyces cerevisiae”
Model-based Analysis of ChIP-Seq (MACS) (2008, neutral): “coupled with genome tiling microarrays (ChIP-chip) [3,4]”
Annotating non-coding regions of the genome (2010, methodology): “For instance, chromatin immunoprecipitation followed by microarray (ChIP–chip”
The evolution of transcriptional regulation in eukaryotes. (2003, background): “Thus, the genetic basis for the expression profile of each gene resides in part within its promoter and in part within the many other segments of the genome that encode specific transcription factors that bind to the promoter.”
Identifying regulatory networks by combinatorial analysis of promoter elements (2001, background): “Experimental results from genome-wide DNA-protein interaction studies 32,38 may help to reﬁne these settings in the future.”
Cell Wall Assembly in Saccharomyces cerevisiae (2006, background): “These data indicate that during the cell cycle, FKS1 expression is regulated by the SBF transcription factor.”
Genomic targets of the human c-Myc protein. (2003, background): “Yeast Rap1 and SBF showed a similar tendency (Iyer et al. 2001; Lieb et al. 2001), suggesting that eukaryotic promoters possess unique structural features.”
S-phase checkpoint proteins Tof1 and Mrc1 form a stable replication-pausing complex (2003, methodology): “This shows that our chromatin immunoprecipitation (ChIP)-chip metho”
From signatures to models: understanding cancer using microarrays (2005, background): “Microarrays, high-throughput chromatin immunoprecipitation 1,2 (ChIP) and tissue microarrays 3 inform us on different perspectives of the molecular mechanisms underlying cellular functions.”
Genome-wide prediction of transcription factor binding sites using an integrated model (2010, neutral): “Chromatin immunoprecipitation (ChIP)-chip [2,3] and, more recently, ChIP-seq have become popular and powerful tools to determine TFBSs at a genome-wide scale [3-5].”